P-selectin chases a butterfly.
作者信息
Wagner D D
出版信息
J Clin Invest. 1995 May;95(5):1955-6. doi: 10.1172/JCI117878.
Abstract
摘要
相似文献
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P-selectin chases a butterfly.P选择素追逐一只蝴蝶。
J Clin Invest. 1995 May;95(5):1955-6. doi: 10.1172/JCI117878.
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Monocyte tethering by P-selectin regulates monocyte chemotactic protein-1 and tumor necrosis factor-alpha secretion. Signal integration and NF-kappa B translocation.P-选择素介导的单核细胞黏附调节单核细胞趋化蛋白-1和肿瘤坏死因子-α的分泌。信号整合与核因子-κB易位。
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Leukocyte interactions mediated by selectins.由选择素介导的白细胞相互作用。
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P-selectin and wound healing.
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Extracellular ATP and ADP activate transcription factor NF-kappa B and induce endothelial cell apoptosis.细胞外ATP和ADP激活转录因子NF-κB并诱导内皮细胞凋亡。
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本文引用的文献
1
Structure, regulation and function of NF-kappa B.核因子-κB的结构、调控与功能
Annu Rev Cell Biol. 1994;10:405-55. doi: 10.1146/annurev.cb.10.110194.002201.
2
DNA-binding proteins. A butterfly flutters by.
Nature. 1995 Jan 26;373(6512):287-8. doi: 10.1038/373287a0.
3
Leukocyte rolling and extravasation are severely compromised in P selectin-deficient mice.在P选择素缺陷型小鼠中，白细胞滚动和外渗严重受损。
Cell. 1993 Aug 13;74(3):541-54. doi: 10.1016/0092-8674(93)80055-j.
4
Monocyte tethering by P-selectin regulates monocyte chemotactic protein-1 and tumor necrosis factor-alpha secretion. Signal integration and NF-kappa B translocation.P-选择素介导的单核细胞黏附调节单核细胞趋化蛋白-1和肿瘤坏死因子-α的分泌。信号整合与核因子-κB易位。
J Clin Invest. 1995 May;95(5):2297-303. doi: 10.1172/JCI117921.
5
Leukocyte-endothelial adhesion molecules.白细胞-内皮细胞黏附分子
Blood. 1994 Oct 1;84(7):2068-101.
6
P-selectin induces the expression of tissue factor on monocytes.P选择素诱导单核细胞上组织因子的表达。
Proc Natl Acad Sci U S A. 1994 Sep 13;91(19):8767-71. doi: 10.1073/pnas.91.19.8767.
7
Expression cloning of a functional glycoprotein ligand for P-selectin.P-选择素功能性糖蛋白配体的表达克隆
Cell. 1993 Dec 17;75(6):1179-86. doi: 10.1016/0092-8674(93)90327-m.
8
Coexpression of GMP-140 and PAF by endothelium stimulated by histamine or thrombin: a juxtacrine system for adhesion and activation of neutrophils.组胺或凝血酶刺激的内皮细胞中GMP-140和PAF的共表达：一种用于中性粒细胞黏附和激活的旁分泌系统。
J Cell Biol. 1991 Oct;115(1):223-34. doi: 10.1083/jcb.115.1.223.
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