Modeling Lou Gehrig's disease in the fruit fly.
作者信息
McCabe E R
机构信息
Department of Pediatrics, University of California School of Medicine, Los Angeles 90024-1752, USA.
出版信息
Proc Natl Acad Sci U S A. 1995 Sep 12;92(19):8533-4. doi: 10.1073/pnas.92.19.8533.
Abstract
摘要
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本文引用的文献
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Motor neurone disease and animal models.运动神经元病与动物模型
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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis.铜锌超氧化物歧化酶基因突变与家族性肌萎缩侧索硬化症相关。
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Bcl-2 inhibition of neural death: decreased generation of reactive oxygen species.Bcl-2对神经细胞死亡的抑制作用：活性氧生成减少。
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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation.表达人类铜锌超氧化物歧化酶突变的小鼠中的运动神经元变性。
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Cu/Zn superoxide dismutase free radicals, and motoneuron disease.铜/锌超氧化物歧化酶自由基与运动神经元病
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Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity.与家族性肌萎缩侧索硬化症相关的突变超氧化物歧化酶1具有显著活性。
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Down-regulation of copper/zinc superoxide dismutase causes apoptotic death in PC12 neuronal cells.铜锌超氧化物歧化酶的下调导致PC12神经细胞凋亡死亡。
Proc Natl Acad Sci U S A. 1994 Jul 5;91(14):6384-7. doi: 10.1073/pnas.91.14.6384.
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