Bansal V K, Brooks M H, York J C, Hano J E
Arch Intern Med. 1979 Jan;139(1):101-2. doi: 10.1001/archinte.139.1.101.
A patient with chronic renal failure developed intractable hypoglycemia after 2 1/2 years of dialysis. Metabolic studies did not suggest that malnutrition, substrate limitation, hormone deficiency, or insulin excess were responsible for the low blood glucose level. Impaired gluconeogenesis may have been an important factor, but studies in this patient suggest that enhanced glucose utilization may also play a role in the pathogenesis of hypoglycemia in renal failure.
一名慢性肾衰竭患者在透析2年半后出现顽固性低血糖。代谢研究表明，营养不良、底物限制、激素缺乏或胰岛素过量并非导致低血糖水平的原因。糖异生受损可能是一个重要因素，但对该患者的研究表明，葡萄糖利用增强在肾衰竭低血糖的发病机制中也可能起作用。
