抗心律失常肽的治疗潜力。细胞耦联作为一个新的抗心律失常靶点。 - Suppr

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超能文献

Therapeutic potential of antiarrhythmic peptides. Cellular coupling as a new antiarrhythmic target.

作者信息

Dhein S, Tudyka T

机构信息

Institut für Pharmakologie, Universität zu Köln, Cologne, Germany.

出版信息

Drugs. 1995 Jun;49(6):851-5. doi: 10.2165/00003495-199549060-00001.

DOI:10.2165/00003495-199549060-00001

PMID:7641601

Abstract

摘要

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本文引用的文献

Junctional membrane permeability : Effects of divalent cations.连接膜通透性：二价阳离子的影响。

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Ultrastructural-functional basis for spontaneous termination of ventricular fibrillation in mammals.哺乳动物心室颤动自发终止的超微结构-功能基础。

J Basic Clin Physiol Pharmacol. 1993 Oct-Dec;4(4):281-90. doi: 10.1515/jbcpp.1993.4.4.281.

Robert Feulgen Prize Lecture. Distribution and role of gap junctions in normal myocardium and human ischaemic heart disease.罗伯特·福尔根奖讲座。缝隙连接在正常心肌和人类缺血性心脏病中的分布及作用。

Histochemistry. 1993 Feb;99(2):105-20. doi: 10.1007/BF00571871.

Comparative study on the proarrhythmic effects of some antiarrhythmic agents.某些抗心律失常药物促心律失常作用的比较研究

Circulation. 1993 Feb;87(2):617-30. doi: 10.1161/01.cir.87.2.617.

Antiarrhythmic drugs and torsade de pointes.抗心律失常药物与尖端扭转型室性心动过速

Eur Heart J. 1993 Nov;14 Suppl H:88-92. doi: 10.1093/eurheartj/14.suppl_h.88.

Initiating reentry: the role of nonuniform anisotropy in small circuits.

J Cardiovasc Electrophysiol. 1994 Feb;5(2):182-209. doi: 10.1111/j.1540-8167.1994.tb01157.x.

Class III antiarrhythmics in overdose. Presenting features and management principles.Ⅲ类抗心律失常药物过量。临床表现及处理原则。

Drug Saf. 1993 Dec;9(6):450-62. doi: 10.2165/00002018-199309060-00008.

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