文献检索，用中文搜 PubMed

Suppr 超能文献

核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Zhao J, Zhao X, Liu W, Duan X, Zhao A, Zhao G, Xue F, Zhang X

Shaanxi Academy of Tradetional Chinese Medicine XI' An.

Zhen Ci Yan Jiu. 1995;20(1):40-3.

In normal control mice (only injected SRBC) after EA the lymphocyte transformation test (LTT, P < 0.05) and interlukin-2 (IL-2) activity (P < 0.05) were significantly increased, but the serum lysozyme (IZM, P < 0.01) was apparantly decreased. 2. After injection of 6-OHDA in mice, the spleen weight, spleen index, thymus weight, thymusindex, spleen lymphocyte population and serum IZM, igG contents were markedly decreased, whereas LTT, IL-2 and plaque forming cell (PFC) were umchanged, In contrast, the thymus lymphocyte population was markedly increased. 3. After EA of mice axotomized sympathetic nerve with 6-OHDA the above mentioned immune parameters deceased by 6-OHDA could be returned to normal or nearing normal levels. However, the unchanged LTT, IL-2 and the increased thymus lymphocyte population could be enhanced further, especially IL-2. These results suggest that peripheral sympathetic nerve may be participation in the EA-mediating immunological responses.

在正常对照小鼠（仅注射绵羊红细胞）中，电针后淋巴细胞转化试验（LTT，P<0.05）和白细胞介素-2（IL-2）活性（P<0.05）显著增加，但血清溶菌酶（IZM，P<0.01）明显降低。2. 给小鼠注射6-羟基多巴胺后，脾脏重量、脾脏指数、胸腺重量、胸腺指数、脾脏淋巴细胞数量以及血清IZM、IgG含量均显著降低，而LTT、IL-2和空斑形成细胞（PFC）无变化，相反，胸腺淋巴细胞数量显著增加。3. 用6-羟基多巴胺切断交感神经的小鼠电针后，上述被6-羟基多巴胺降低的免疫参数可恢复到正常或接近正常水平。然而，未变化的LTT、IL-2以及增加的胸腺淋巴细胞数量可进一步增强，尤其是IL-2。这些结果表明外周交感神经可能参与电针介导的免疫反应。

Zhao J, Zhao X, Liu W, Duan X, Zhao A, Zhao G, Xue F, Zhang X

Shaanxi Academy of Tradetional Chinese Medicine XI' An.

Zhen Ci Yan Jiu. 1995;20(1):40-3.

In normal control mice (only injected SRBC) after EA the lymphocyte transformation test (LTT, P < 0.05) and interlukin-2 (IL-2) activity (P < 0.05) were significantly increased, but the serum lysozyme (IZM, P < 0.01) was apparantly decreased. 2. After injection of 6-OHDA in mice, the spleen weight, spleen index, thymus weight, thymusindex, spleen lymphocyte population and serum IZM, igG contents were markedly decreased, whereas LTT, IL-2 and plaque forming cell (PFC) were umchanged, In contrast, the thymus lymphocyte population was markedly increased. 3. After EA of mice axotomized sympathetic nerve with 6-OHDA the above mentioned immune parameters deceased by 6-OHDA could be returned to normal or nearing normal levels. However, the unchanged LTT, IL-2 and the increased thymus lymphocyte population could be enhanced further, especially IL-2. These results suggest that peripheral sympathetic nerve may be participation in the EA-mediating immunological responses.

在正常对照小鼠（仅注射绵羊红细胞）中，电针后淋巴细胞转化试验（LTT，P<0.05）和白细胞介素-2（IL-2）活性（P<0.05）显著增加，但血清溶菌酶（IZM，P<0.01）明显降低。2. 给小鼠注射6-羟基多巴胺后，脾脏重量、脾脏指数、胸腺重量、胸腺指数、脾脏淋巴细胞数量以及血清IZM、IgG含量均显著降低，而LTT、IL-2和空斑形成细胞（PFC）无变化，相反，胸腺淋巴细胞数量显著增加。3. 用6-羟基多巴胺切断交感神经的小鼠电针后，上述被6-羟基多巴胺降低的免疫参数可恢复到正常或接近正常水平。然而，未变化的LTT、IL-2以及增加的胸腺淋巴细胞数量可进一步增强，尤其是IL-2。这些结果表明外周交感神经可能参与电针介导的免疫反应。