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Proteolysis of IGFBP-3 may be a common regulatory mechanism of IGF action in vivo.

作者信息

Lamson G, Giudice L C, Cohen P, Liu F, Gargosky S, Müller H L, Oh Y, Wilson K F, Hintz R L, Rosenfeld R G

机构信息

Department of Pediatrics, Stanford University School of Medicine, CA.

出版信息

Growth Regul. 1993 Mar;3(1):91-5.

PMID:7683546
Abstract
摘要

相似文献

1
Proteolysis of IGFBP-3 may be a common regulatory mechanism of IGF action in vivo.胰岛素样生长因子结合蛋白-3(IGFBP-3)的蛋白水解作用可能是体内胰岛素样生长因子(IGF)作用的一种常见调节机制。
Growth Regul. 1993 Mar;3(1):91-5.
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Matrix metalloproteinase-7 degrades all insulin-like growth factor binding proteins and facilitates insulin-like growth factor bioavailability.基质金属蛋白酶-7可降解所有胰岛素样生长因子结合蛋白并促进胰岛素样生长因子的生物利用度。
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引用本文的文献

1
Insulin-like growth factors and their binding proteins: Potential relevance to reproductive physiology.胰岛素样生长因子及其结合蛋白:与生殖生理学的潜在关联。
Reprod Med Biol. 2003 Mar 25;2(1):1-24. doi: 10.1046/j.1445-5781.2003.00016.x. eCollection 2003 Mar.
2
Damage control mechanisms in articular cartilage: the role of the insulin-like growth factor I axis.关节软骨中的损伤控制机制:胰岛素样生长因子I轴的作用。
Iowa Orthop J. 2000;20:1-10.
3
Postsynthetic regulation of insulin-like growth factor-binding protein-3 by MCF-7 human breast cancer cells in culture.
培养的MCF-7人乳腺癌细胞对胰岛素样生长因子结合蛋白-3的合成后调控
Breast Cancer Res Treat. 1996;39(2):187-96. doi: 10.1007/BF01806185.