P物质诱导的大鼠膝关节炎症由神经激肽1（NK1）受体介导。 - Suppr

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超能文献

Substance P-induced inflammation in the rat knee joint is mediated by neurokinin 1 (NK1) receptors.

作者信息

Lam F Y, Ferrell W R, Scott D T

机构信息

Institute of Physiology, University of Glasgow, UK.

出版信息

Regul Pept. 1993 Jul 2;46(1-2):198-201. doi: 10.1016/0167-0115(93)90032-4.

DOI:10.1016/0167-0115(93)90032-4

PMID:7692489

Abstract

摘要

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Substance P-induced inflammation in the rat knee joint is mediated by neurokinin 1 (NK1) receptors.P物质诱导的大鼠膝关节炎症由神经激肽1（NK1）受体介导。

Regul Pept. 1993 Jul 2;46(1-2):198-201. doi: 10.1016/0167-0115(93)90032-4.

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Characterization of tachykinin receptors mediating plasma extravasation and vasodilatation in normal and acutely inflamed knee joints of the rat.介导大鼠正常和急性炎症膝关节血浆外渗及血管舒张的速激肽受体的特性研究

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Effects of interactions of naturally-occurring neuropeptides on blood flow in the rat knee joint.天然存在的神经肽相互作用对大鼠膝关节血流的影响。

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NK1 receptors mediate tachykinin-induced plasma extravasation in the rat knee joint.NK1受体介导速激肽诱导的大鼠膝关节血浆外渗。

Agents Actions. 1993 Nov;40(3-4):171-5. doi: 10.1007/BF01984057.

Effect of scyliorhinin I and synthetic scyliorhinin I derivatives at mammalian tachykinin NK1, NK2 and NK3 receptors.鲨肌素I及合成鲨肌素I衍生物对哺乳动物速激肽NK1、NK2和NK3受体的作用。

Eur J Pharmacol. 1993 Dec 7;250(2):311-6. doi: 10.1016/0014-2999(93)90396-y.

The role of spinal neurokinin-2 receptors in the processing of nociceptive information from the joint and in the generation and maintenance of inflammation-evoked hyperexcitability of dorsal horn neurons in the rat.脊髓神经激肽-2受体在处理来自关节的伤害性信息以及在大鼠背角神经元炎症诱发的兴奋性过高的产生和维持中的作用。

Eur J Neurosci. 1996 Feb;8(2):249-60. doi: 10.1111/j.1460-9568.1996.tb01209.x.

NK1 receptors mediate neurogenic inflammatory increase in blood flow in rat airways.NK1受体介导大鼠气道中神经源性炎症引起的血流增加。

J Appl Physiol (1985). 1993 May;74(5):2462-8. doi: 10.1152/jappl.1993.74.5.2462.

A non-peptide NK1-receptor antagonist, RP 67580, inhibits neurogenic inflammation postsynaptically.一种非肽类NK1受体拮抗剂RP 67580，可在突触后抑制神经源性炎症。

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Tachykinin regulation of basal synovial blood flow.速激肽对滑膜基础血流的调节。

Br J Pharmacol. 1997 May;121(1):29-34. doi: 10.1038/sj.bjp.0701095.

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Substance P and capsaicin-induced mechanical hyperalgesia in the rat knee joint; the involvement of bradykinin B1 and B2 receptors.P物质和辣椒素诱导的大鼠膝关节机械性痛觉过敏；缓激肽B1和B2受体的作用

Br J Pharmacol. 1996 Aug;118(8):2206-12. doi: 10.1111/j.1476-5381.1996.tb15664.x.

Br J Pharmacol. 1996 Aug;118(8):2107-14. doi: 10.1111/j.1476-5381.1996.tb15650.x.

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