Wachsberg R H, Needleman L, Wilson D J
Department of Radiology, University Hospital, Newark, New Jersey 07103.
J Clin Ultrasound. 1995 Jan;23(1):3-15. doi: 10.1002/jcu.1870230103.
The literature indicates that Doppler demonstration of pulsatile flow in the portal vein suggests heart disease, and that retrograde transsinusoidal transmission of atrial pulsations is the mechanism. We noninvasively investigated portal vein pulsatility (PVP) in normal subjects and in patients with cardiac and liver disease, and performed invasive studies in cirrhotic humans and normal pigs. We found that accentuated PVP occurred in some normal subjects and in some patients with cirrhosis, and that mechanisms other than transsinusoidal transmission of atrial pulsations contributed to PVP. Determinants of PVP may include pulsatile portal inflow, transmission of pulsations from the vena cava (IVC) and location of the Doppler sample volume relative to the IVC.
文献表明，门静脉搏动血流的多普勒显示提示心脏病，而心房搏动的逆行经窦状隙传导是其机制。我们对正常受试者以及患有心脏和肝脏疾病的患者进行了门静脉搏动性（PVP）的无创研究，并对肝硬化患者和正常猪进行了有创研究。我们发现，一些正常受试者和一些肝硬化患者出现了PVP增强，并且除了心房搏动的经窦状隙传导之外的其他机制也导致了PVP。PVP的决定因素可能包括搏动性门静脉血流、来自腔静脉（IVC）的搏动传导以及多普勒取样容积相对于IVC的位置。
