Henry-Le Bras F, Fischer C, Nighoghossian N, Salord F, Trouillas P, Mauguière F
Service de neurologie fonctionnelle et d'épileptologie, hôpital neurologique, Lyon, France.
Neurophysiol Clin. 1994 Dec;24(6):399-412. doi: 10.1016/s0987-7053(05)80073-5.
Brainstem auditory evoked potentials (BAEPs) and middle-latency auditory evoked potentials (MLAEPs) have been recorded in 67 patients who had a stroke in well-defined territories of the vertebral and basilar arteries. Either CT scan or MRI have been performed in all cases. BAEPs were abnormal in 41/67 patients and MLAEPs were abnormal in 25/39 patients. BAEPs abnormalities were either bilateral (29/41 cases) or unilateral (12/41 cases). All components of BAEPS were unilaterally absent in four cases and bilaterally in one case. Pa component of MLAEPs was unilaterally delayed or reduced in five cases and bilaterally in 20 cases. Considering the topography of the infarct as shown by CT scan or MRI: medulla oblongata (13 cases): BAEPSs were normal in nine cases; pons (24 cases): BAEPs were abnormal in 16 cases; MLAEPs were abnormal in ten of 15 patients whose BAEPs were abnormal as well; mesencephalon (seven cases): BAEPs were abnormal in only two cases, and MLAEPs were abnormal in two cases one of which BAEPs were normal; in patients with diffuse infarctions either BAEPs or MLAEPs or both were abnormal in all cases. Stimulation of the ear ipsilateral to the lesion disclosed more BAEPs or MLAEPs abnormalities than stimulation of the contralateral ear.
对67例在椎动脉和基底动脉明确区域发生中风的患者记录了脑干听觉诱发电位(BAEP)和中潜伏期听觉诱发电位(MLAEP)。所有病例均进行了CT扫描或MRI检查。67例患者中41例BAEP异常,39例患者中25例MLAEP异常。BAEP异常为双侧(41例中的29例)或单侧(41例中的12例)。4例BAEP所有成分单侧缺失,1例双侧缺失。MLAEP的Pa成分5例单侧延迟或降低,20例双侧延迟或降低。根据CT扫描或MRI显示的梗死部位:延髓(13例):9例BAEP正常;脑桥(24例):16例BAEP异常;15例BAEP异常的患者中有10例MLAEP异常;中脑(7例):仅2例BAEP异常,2例MLAEP异常,其中1例BAEP正常;在弥漫性梗死患者中,所有病例的BAEP或MLAEP或两者均异常。刺激病变同侧耳朵比刺激对侧耳朵发现更多的BAEP或MLAEP异常。