胰岛素依赖型糖尿病的预防。将胰岛素注入细胞因子网络。 - Suppr | 超能文献

The prevention of IDDM. Injecting insulin into the cytokine network.

作者信息

Gladstone P, Nepom G T

出版信息

Diabetes. 1995 Jul;44(7):859-62. doi: 10.2337/diab.44.7.859.

DOI:10.2337/diab.44.7.859

Abstract

摘要

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

文档翻译

学术文献翻译模型，支持多种主流文档格式。

相似文献

1

The prevention of IDDM. Injecting insulin into the cytokine network.胰岛素依赖型糖尿病的预防。将胰岛素注入细胞因子网络。

Diabetes. 1995 Jul;44(7):859-62. doi: 10.2337/diab.44.7.859.

2

Immunoregulatory and cytokine imbalances in the pathogenesis of IDDM. Therapeutic intervention by immunostimulation?胰岛素依赖型糖尿病发病机制中的免疫调节与细胞因子失衡。免疫刺激的治疗干预？

Diabetes. 1994 May;43(5):613-21. doi: 10.2337/diab.43.5.613.

3

Molecular role of TGF-beta, secreted from a new type of CD4+ suppressor T cell, NY4.2, in the prevention of autoimmune IDDM in NOD mice.新型CD4+抑制性T细胞NY4.2分泌的转化生长因子β（TGF-β）在预防非肥胖糖尿病（NOD）小鼠自身免疫性胰岛素依赖型糖尿病（IDDM）中的分子作用。

J Autoimmun. 1997 Jun;10(3):299-307. doi: 10.1006/jaut.1997.0137.

4

Th1 to Th2 cytokine shifts in nonobese diabetic mice: sometimes an outcome, rather than the cause, of diabetes resistance elicited by immunostimulation.非肥胖糖尿病小鼠中Th1向Th2细胞因子的转变：有时是免疫刺激引发的糖尿病抵抗的结果，而非原因。

J Immunol. 2001 Jan 15;166(2):1352-9. doi: 10.4049/jimmunol.166.2.1352.

5

Immunological and metabolic effects of prophylactic insulin therapy in the NOD-scid/scid adoptive transfer model of IDDM.

Diabetes. 1996 Feb;45(2):205-8. doi: 10.2337/diab.45.2.205.

6

alpha/beta-T cell receptor (TCR)+CD4-CD8- (NKT) thymocytes prevent insulin-dependent diabetes mellitus in nonobese diabetic (NOD)/Lt mice by the influence of interleukin (IL)-4 and/or IL-10.α/β-T细胞受体（TCR）+CD4-CD8-（自然杀伤T细胞，NKT）胸腺细胞通过白细胞介素（IL）-4和/或IL-10的作用预防非肥胖糖尿病（NOD）/Lt小鼠的胰岛素依赖型糖尿病。

J Exp Med. 1998 Apr 6;187(7):1047-56. doi: 10.1084/jem.187.7.1047.

7

Cytokine- and costimulation-mediated therapy of IDDM.细胞因子和共刺激介导的胰岛素依赖型糖尿病治疗

Crit Rev Immunol. 1997;17(5-6):537-44.

8

Insulin-like growth factor-1 (IGF-1)-derived peptide protects against diabetes in NOD mice.

Autoimmunity. 2004 Sep-Nov;37(6-7):481-7. doi: 10.1080/08916930400001909.

9

Cytokines activate caspase-3 in insulinoma cells of diabetes-prone NOD mice directly and via upregulation of Fas.细胞因子可直接激活易患糖尿病的非肥胖糖尿病（NOD）小鼠胰岛素瘤细胞中的半胱天冬酶-3，并通过上调Fas来激活。

J Autoimmun. 2004 Dec;23(4):301-9. doi: 10.1016/j.jaut.2004.09.006.

10

Inhibition of autoimmune diabetes in nonobese diabetic mice by transgenic restoration of H2-E MHC class II expression: additive, but unequal, involvement of multiple APC subtypes.通过转基因恢复H2-E MHC II类分子表达抑制非肥胖糖尿病小鼠的自身免疫性糖尿病：多种抗原呈递细胞亚型的累加但不等同参与。

J Immunol. 2001 Aug 15;167(4):2404-10. doi: 10.4049/jimmunol.167.4.2404.

引用本文的文献

1

Concurrent minimal change nephrotic syndrome and type 1 diabetes mellitus in an adult Japanese woman: a case report.成人日本女性同时患有微小病变性肾病综合征和 1 型糖尿病：病例报告。

BMC Nephrol. 2020 Sep 23;21(1):410. doi: 10.1186/s12882-020-02071-6.

2

Antigen-based immune therapeutics for type 1 diabetes: magic bullets or ordinary blanks?用于1型糖尿病的基于抗原的免疫疗法：神奇子弹还是普通空白？

Clin Dev Immunol. 2011;2011:286248. doi: 10.1155/2011/286248. Epub 2011 May 14.

3

Metabolically inactive insulin analog prevents type I diabetes in prediabetic NOD mice.代谢惰性胰岛素类似物可预防糖尿病前期非肥胖糖尿病（NOD）小鼠患I型糖尿病。

J Clin Invest. 1997 Sep 15;100(6):1344-8. doi: 10.1172/JCI119654.