Yashar A, Loder R T, Hensinger R N
Section of Orthopaedic Surgery, University of Michigan, School of Medicine, Ann Arbor, USA.
J Pediatr Orthop. 1995 May-Jun;15(3):298-301. doi: 10.1097/01241398-199505000-00006.
We determined the skeletal age in 26 children (12 girls, 14 boys) with Osgood-Schlatter disease using knee radiographs. The average chronological age was 13.2 +/- 2.2 years, and the average bone age was 13.1 +/- 2.3 years (p > 0.01). All but one of the skeletal ages fell within the normal range. Skeletal maturation in children with Osgood-Schlatter disease is thus normal. With normal skeletal maturation and normal physes histologically (previously shown), it is unlikely that an abnormality of physeal development or structure is the etiology of Osgood-Schlatter disease. Rather, it is most likely a result of tensile stresses on the tibial tuberosity. This is in contrast to slipped capital femoral epiphysis, another common disorder affecting children of the same ages as those with Osgood-Schlatter disease, in which physeal abnormalities and skeletal maturation anomalies do occur.
我们使用膝关节X线片测定了26例患有奥斯古德-施拉特病的儿童（12名女孩，14名男孩）的骨骼年龄。平均实际年龄为13.2±2.2岁，平均骨龄为13.1±2.3岁（p>0.01）。除1例骨骼年龄外，其余均在正常范围内。因此，患有奥斯古德-施拉特病的儿童骨骼成熟正常。鉴于骨骼成熟正常且组织学上骺板正常（先前已证实），骺板发育或结构异常不太可能是奥斯古德-施拉特病的病因。相反，它很可能是胫骨结节受到拉伸应力的结果。这与股骨头骨骺滑脱相反，股骨头骨骺滑脱是另一种影响与患有奥斯古德-施拉特病儿童相同年龄段儿童的常见疾病，其中确实会出现骺板异常和骨骼成熟异常。
