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Arch Dis Child Fetal Neonatal Ed. 1995 May;72(3):F147-50. doi: 10.1136/fn.72.3.f147.
2
[Brain protection--update].
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3
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4
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5
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6
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7
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9
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3
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5
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本文引用的文献
1
Developmental switch in the expression of NMDA receptors occurs in vivo and in vitro.
Neuron. 1993 Feb;10(2):267-78. doi: 10.1016/0896-6273(93)90317-k.
2
Glycine site of the excitatory amino acid N-methyl-D-aspartate receptor in neonatal and adult brain.
Arch Dis Child. 1993 Aug;69(2):212-5. doi: 10.1136/adc.69.2.212.
3
Cloning, functional coexpression, and pharmacological characterisation of human cDNAs encoding NMDA receptor NR1 and NR2A subunits.
J Neurochem. 1994 Jun;62(6):2091-8. doi: 10.1046/j.1471-4159.1994.62062091.x.
4
Regional patterns of cholinergic and glutamate activity in the developing and aging human brain.
Brain Res Dev Brain Res. 1993 Jul 16;74(1):73-82. doi: 10.1016/0165-3806(93)90085-o.
5
Nitric oxide increases interstitial excitatory amino acid release in the rat dorsomedial medulla oblongata.
Neurosci Lett. 1993 Mar 19;151(2):126-9. doi: 10.1016/0304-3940(93)90002-3.
6
Postnatal changes in N-methyl-D-aspartate receptor binding and stimulation by glutamate and glycine of [3H]-MK-801 binding in human temporal cortex.
Br J Pharmacol. 1993 Apr;108(4):1143-9. doi: 10.1111/j.1476-5381.1993.tb13518.x.
7
Ifenprodil discriminates subtypes of the N-methyl-D-aspartate receptor: selectivity and mechanisms at recombinant heteromeric receptors.
Mol Pharmacol. 1993 Oct;44(4):851-9.
8
The high affinity uptake system for excitatory amino acids in the brain.
Prog Neurobiol. 1994 Nov;44(4):377-96. doi: 10.1016/0301-0082(94)90033-7.
9
The anticonvulsant and behavioural profile of L-687,414, a partial agonist acting at the glycine modulatory site on the N-methyl-D-aspartate (NMDA) receptor complex.
Br J Pharmacol. 1994 Nov;113(3):729-36. doi: 10.1111/j.1476-5381.1994.tb17054.x.
10
Therapeutic potential of excitatory amino acid antagonists: channel blockers and 2,3-benzodiazepines.
Trends Pharmacol Sci. 1993 Sep;14(9):325-31. doi: 10.1016/0165-6147(93)90005-5.
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