de la Balze F A
Rev Iber Endocrinol. 1976 May-Jun;23(135):225-43.
Clinical and experimental studies have suggested the author the possibility that an androgenic insufficiency, during embryogenesis or the perinatal period, may be a determinant factor in the pathogenesis of the anomalies of homosexual behavior. In the author's opinion, the androgenic anomalies seen in the genuine male homosexual have no pathogenic significance, but are a repetition of what occured during embryogenesis of the perinatal period, at the time when these anomalies had a pathogenic role.
临床和实验研究向作者表明,在胚胎发育或围产期雄激素不足可能是同性恋行为异常发病机制中的一个决定性因素。作者认为,真性男性同性恋者中所见的雄激素异常没有致病意义,而是在围产期胚胎发育期间所发生情况的重复,当时这些异常具有致病作用。