Martinez Moreno J L
Clinique de Chirurgie Orthopédique et Traumatologique C/ Santisima Trinidad, Madrid, Espagne.
Rev Chir Orthop Reparatrice Appar Mot. 1994;80(3):239-45.
A prospective arthroscopic study on 140 knees to discover the etiopathogenic mechanism responsible for idiopathic patellar syndrome (IPS) was carried out.
Patients were grouped into two categories: One group with idiopathic patellar syndrome (76 patients, 53 male and 23 female), the other group (control group) presenting no patellar pathology in clinical, radiological and arthroscopic examinations (64 patients, 55 male and 9 female). The author analyzed the statistical differences using 9 clinical and morphometric parameters in the IPS group and the control group. The morphometric index PTO/LNPL (PTO: distance between the medial upper end of the tibia and the inferior border of the pes anserinus. LNPL: width of medial tibial surface) seems to be a valid reference index (p = 0) for the diagnosis of IPSI caused by internal rotatory muscle deficiency.
The X2 test showed significative correlation (p < 0.05) between sex and IPS. The incidence of IPS was 1/1 in males and 2.5/1 in females. The PTO/LNPL value in this syndrome was higher than 1.1. No patient in the IPS group had spontaneous recovery, nevertheless, 86.8 per cent of patients has a good result after 6 months of physical therapy of the internal rotatory muscles.
The high distance between the medial upper end of the tibia and the inferior border of the pes anserinus in knees with IPS as well as the good results obtained by physical therapy of the internal rotatory muscles leads the author to suggest a new etiopathogenic mechanism for IPS: some idiopathic patellar syndromes may be caused by an isolated internal rotatory muscle deficiency of the leg.
The author concludes with two therapeutic suggestions: 1. physical therapy of the internal rotatory muscles should be used in patellar syndromes caused by deficiency of internal rotatory muscles. 2. Methods which sacrifice internal rotatory muscles in knee ligament surgery should be avoided as these could lead to a possible patellar syndrome by deficiency of internal rotatory muscles.
开展了一项针对140个膝关节的前瞻性关节镜研究,以探寻特发性髌股综合征(IPS)的发病机制。
患者分为两类:一类为特发性髌股综合征患者(76例,男53例,女23例),另一类(对照组)在临床、放射学和关节镜检查中均无髌股病变(64例,男55例,女9例)。作者分析了IPS组和对照组9项临床和形态学参数的统计学差异。形态学指标PTO/LNPL(PTO:胫骨内侧上端与鹅足肌腱下边界之间的距离。LNPL:胫骨内侧表面宽度)似乎是诊断由内旋肌缺乏引起的IPSI的有效参考指标(p = 0)。
X²检验显示性别与IPS之间存在显著相关性(p < 0.05)。IPS的发病率在男性中为1/1,在女性中为2.5/1。该综合征的PTO/LNPL值高于1.1。IPS组中无患者自发恢复,然而,86.8%的患者在对内旋肌进行6个月的物理治疗后取得了良好效果。
IPS膝关节中胫骨内侧上端与鹅足肌腱下边界之间的距离较大,以及对内旋肌进行物理治疗取得的良好效果,使作者提出了一种新的IPS发病机制:一些特发性髌股综合征可能由腿部孤立的内旋肌缺乏引起。
作者得出两条治疗建议:1. 对于由内旋肌缺乏引起的髌股综合征,应采用对内旋肌的物理治疗。2. 应避免在膝关节韧带手术中采用牺牲内旋肌的方法,因为这些方法可能因内旋肌缺乏而导致髌股综合征。