自体骨髓移植后发生的吉兰-巴雷综合征。
Guillain-Barre syndrome following autologous bone marrow transplantation.
作者信息
Mudad R, Hussein A, Peters W P
机构信息
Department of Hematology-Oncology, Duke University Medical Center, Durham, North Carolina 27710.
出版信息
Am J Clin Oncol. 1995 Apr;18(2):167-9. doi: 10.1097/00000421-199504000-00016.
The pathogenesis of the Guillain-Barre syndrome (GBS) is felt to involve alterations in cellular and humoral immune responses. We report the case of a 44-year-old female with stage IV breast cancer who developed GBS 2 days after receiving high dose chemotherapy with bone marrow and peripheral progenitor cell support, and granulocyte-macrophage colony stimulating factor (GM-CSF). Her clinical status stabilized with plasmapheresis. This report lends further support to hypotheses regarding cellular immune alterations in the pathogenesis of GBS.
格林-巴利综合征（GBS）的发病机制被认为涉及细胞免疫和体液免疫反应的改变。我们报告一例44岁患有IV期乳腺癌的女性病例，该患者在接受高剂量化疗并伴有骨髓和外周祖细胞支持以及粒细胞-巨噬细胞集落刺激因子（GM-CSF）治疗2天后发生了GBS。她的临床状况通过血浆置换得以稳定。本报告进一步支持了关于GBS发病机制中细胞免疫改变的假说。
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