分枝杆菌脂阿拉伯甘露聚糖在巨噬细胞系中诱导一氧化氮和肿瘤坏死因子α的产生：牛磺酸氯胺的下调作用
Mycobacterial lipoarabinomannan induces nitric oxide and tumor necrosis factor alpha production in a macrophage cell line: down regulation by taurine chloramine.
作者信息
Schuller-Levis G B, Levis W R, Ammazzalorso M, Nosrati A, Park E
机构信息
Staten Island University Hospital, New York 10305.
出版信息
Infect Immun. 1994 Oct;62(10):4671-4. doi: 10.1128/iai.62.10.4671-4674.1994.
Abstract
Avirulent mycobacterium H37Ra lipoarabinomannan (LAM) elicited nitric oxide (NO) and tumor necrosis factor alpha in a dose-dependent manner in a murine macrophage cell line, RAW 264.7 cells. H37Ra LAM and recombinant gamma interferon were highly synergistic for NO production. The production of NO and the release of tumor necrosis factor alpha stimulated by H37Ra LAM plus recombinant gamma interferon in RAW 264.7 cells are inhibited by taurine chloramine.
摘要
无毒力的结核分枝杆菌H37Ra脂阿拉伯甘露聚糖（LAM）在小鼠巨噬细胞系RAW 264.7细胞中以剂量依赖方式诱导产生一氧化氮（NO）和肿瘤坏死因子α。H37Ra LAM与重组γ干扰素在产生NO方面具有高度协同作用。RAW 264.7细胞中由H37Ra LAM加重组γ干扰素刺激产生的NO及肿瘤坏死因子α的释放受到氯胺酮的抑制。
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