Hastings H, Graham T J
Department of Orthopaedic Surgery, Indiana University Medical Center, Indianapolis.
Hand Clin. 1994 Aug;10(3):417-37.
Successful treatment of HO about the forearm and elbow relies on a working understanding of the risk factors, the pathophysiology and pathoanatomy, and the potential role for reconstructive procedures. These elements must be combined with a certain degree of flexibility in the approach to patients with a wide range of individual needs. Class I HO should be managed primarily with close observation, serial radiographs, and appropriate physical therapy regimens. The temporal relationship between the insult and the appearance of HO may modify the approach. When HO is noted within the first 6 weeks, use of anti-inflammatory agents is recommended; if the patient has developed limiting ectopic bone in the past, consideration should be given to a single dose of radiotherapy. In the 6-week to 3-month period, therapy is conducted to maintain full motion and an anti-inflammatory agent continued or started. We have not observed initial HO appearance after the third month. Class IIA HO can involve the anterior or posterior aspects of the elbow joint, or both. These groups are further divided into those limited by soft tissue (muscle and capsular contracture) and those blocked by bone (coronoid extension, humeroradial, humeroulnar, blocked olecranon fossa). The anterior group limited by soft tissue is addressed by capsulotomy, releases, and lengthenings. This group requires careful neurolysis and protection of vascular structures. For anterior bony bridges, resection is combined with capsulotomy. The location of the forearm "insertion" site of the new bone dictates alternative procedures such as interposition or radial head resection. The condition of the joint is usually preserved in these cases, but arthroplasty must always be considered when injury has led to joint derangement. Posteriorly, limitations in motion are caused by a contracted scarred triceps, capsular contracture, or bony impingement and synostosis. Treatment requires posterior capsular release and triceps tenolysis. Bridging bone is excised, the olecranon partially excised, and the olecranon fossa reestablished. Attempts should be made to preserve the fat pad of the olecranon fossa, which can act as an effective interposition material. Although characterized by limited pronosupination, class IIB HO can be located in any of the six distinct anatomic sites previously outlined. Simple resection, with or without interposition, is useful for a majority of the HO that is coincident with the interosseous membrane, but the areas at the proximal and distal extent of the forearm may demand special procedures to restore motion.(ABSTRACT TRUNCATED AT 400 WORDS)
成功治疗前臂和肘部的异位骨化依赖于对风险因素、病理生理学和病理解剖学以及重建手术潜在作用的有效理解。这些要素必须与针对具有广泛个体需求患者的一定程度的灵活性相结合。I类异位骨化主要应通过密切观察、系列X线片和适当的物理治疗方案来处理。损伤与异位骨化出现之间的时间关系可能会改变处理方法。如果在最初6周内发现异位骨化,建议使用抗炎药;如果患者过去曾出现限制活动的异位骨,则应考虑给予单次放射治疗剂量。在6周龄至3个月期间,进行治疗以维持完全活动,并继续或开始使用抗炎药。我们未观察到3个月后出现初始异位骨化的情况。IIA类异位骨化可累及肘关节的前方或后方,或两者皆有。这些组进一步分为受软组织(肌肉和关节囊挛缩)限制的和受骨阻挡(冠状突延长、肱桡、肱尺、鹰嘴窝受阻)的。受软组织限制的前方组通过关节囊切开、松解和延长来处理。该组需要仔细进行神经松解并保护血管结构。对于前方骨桥,切除与关节囊切开相结合。新骨在前臂“插入”部位的位置决定了诸如植入或桡骨头切除等替代手术。在这些情况下,关节状况通常得以保留,但当损伤导致关节紊乱时,必须始终考虑关节成形术。在后方,活动受限是由三头肌挛缩瘢痕、关节囊挛缩或骨撞击和骨桥形成引起的。治疗需要进行后方关节囊松解和三头肌松解。切除桥接骨,部分切除鹰嘴,并重建鹰嘴窝。应尝试保留鹰嘴窝的脂肪垫,其可作为有效的植入材料。尽管以旋前旋后受限为特征,但IIB类异位骨化可位于先前概述的六个不同解剖部位中的任何一个。对于大多数与骨间膜相关的异位骨化,简单切除(有或无植入)是有用的,但前臂近端和远端范围的区域可能需要特殊手术来恢复活动。(摘要截断于400字)