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Lack of a detectable effect of capsid proteins on the cell-dependent activity of parvovirus MVMp promoters.

作者信息

Spegelaere P, Cornelis J J, Tuynder M, Rommelaere J

机构信息

Department of Molecular Biology, Université Libre de Bruxelles, Rhode St. Genese, Belgium.

出版信息

Res Virol. 1994 Jan-Feb;145(1):5-12. doi: 10.1016/s0923-2516(07)80001-x.

DOI:10.1016/s0923-2516(07)80001-x
PMID:8023015
Abstract

Deletion of upstream elements from the early P4 promoter of parvovirus MVM (minute virus of mice, prototype strain MVMp) has a differential effect on its activity, depending on the host cell considered. This indicates that upstream motifs participate in the control of promoter P4 functioning and are responsive to factors which are, at least in part, cell-type specific. In contrast with other viral systems, the capsid proteins of MVMp had no detectable effect on gene expression driven by either the early P4 or late P38 promoter of MVMp in permissive and non-permissive cells.

摘要

相似文献

1
Lack of a detectable effect of capsid proteins on the cell-dependent activity of parvovirus MVMp promoters.
Res Virol. 1994 Jan-Feb;145(1):5-12. doi: 10.1016/s0923-2516(07)80001-x.
2
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J Virol. 2005 Sep;79(17):11280-90. doi: 10.1128/JVI.79.17.11280-11290.2005.

引用本文的文献

1
DNA unwinding functions of minute virus of mice NS1 protein are modulated specifically by the lambda isoform of protein kinase C.小鼠微小病毒NS1蛋白的DNA解旋功能受到蛋白激酶C的λ亚型的特异性调节。
J Virol. 1999 Sep;73(9):7410-20. doi: 10.1128/JVI.73.9.7410-7420.1999.
2
Inhibition of parvovirus minute virus of mice replication by a peptide involved in the oligomerization of nonstructural protein NS1.一种参与非结构蛋白NS1寡聚化的肽对小鼠细小病毒复制的抑制作用。
J Virol. 1997 Oct;71(10):7393-403. doi: 10.1128/JVI.71.10.7393-7403.1997.
3
Transactivation of a cellular promoter by the NS1 protein of the parvovirus minute virus of mice through a putative hormone-responsive element.
小鼠细小病毒的NS1蛋白通过一个假定的激素反应元件对细胞启动子进行反式激活。
J Virol. 1996 Apr;70(4):2369-77. doi: 10.1128/JVI.70.4.2369-2377.1996.
4
NF-Y controls transcription of the minute virus of mice P4 promoter through interaction with an unusual binding site.核因子Y通过与一个异常结合位点相互作用来控制小鼠微小病毒P4启动子的转录。
J Virol. 1995 Jan;69(1):239-46. doi: 10.1128/JVI.69.1.239-246.1995.