Decaux G, Namias B, Soupart A
Service de Médecine interne, Hôpital Erasme, Bruxelles.
Rev Med Brux. 1994 Mar-Apr;15(2):80-5.
Hyponatraemia secondary to inappropriate secretion of antidiuretic hormone (SIADH) induces some biological modifications which are relatively characteristic of this condition. The disturbed acid-base equilibrium and the mechanism involved in the high urea and uric acid clearances observed in SIADH are discussed.
抗利尿激素分泌不当综合征(SIADH)继发的低钠血症会引发一些相对具有该病症特征的生物学改变。本文讨论了SIADH中酸碱平衡紊乱以及高尿素和尿酸清除率所涉及的机制。