Campbell S B, Macfarlane D J, Fleming S J, Khafagi F A
Department of Renal Medicine, Royal Brisbane Hospital, Australia.
Clin Nucl Med. 1994 Mar;19(3):207-11. doi: 10.1097/00003072-199403000-00008.
A case of milk-alkali syndrome is described in a 34-year-old man taking an over-the-counter antacid preparation for gastroesophageal reflux. A Tc-99m MDP bone scan performed in the initial investigation of the hypercalcemia was markedly abnormal with a "metabolic" pattern of tracer uptake similar to that seen in hyperparathyroidism and humoral hypercalcemia. Following withdrawal of the antacid and calcium, the bone scan appearance returned to normal, as did the biochemical markers of his disease.
本文描述了一例34岁男性的乳碱综合征病例，该患者因胃食管反流服用非处方抗酸剂。在对高钙血症进行初步检查时进行的锝-99m亚甲基二膦酸盐（Tc-99m MDP）骨扫描结果明显异常，示踪剂摄取呈“代谢性”模式，类似于甲状旁腺功能亢进和体液性高钙血症所见。停用抗酸剂和钙剂后，骨扫描表现恢复正常，其疾病的生化指标也恢复正常。
