Gutiérrez Ganzarain A, Playán Usón J, Rubio Aranda E, Castro Ascaso M C, Herrero García T, Acha J
Servicio de Medicina Interna, Hospital Servet, Zaragoza.
An Med Interna. 1993 Dec;10(12):583-6.
Mechanism tobacco favours arteriosclerosis aren't clear. Hyperglycemia in smokers would be help factor. On value relationship between tobacco, fasting plasma glucose and hemoglobin A1c in a coal-miner working population, made up of 1,379 males, 38 years old average. We find meaning difference in hemoglobin A1c and fasting plasma glucose values between smokers and non-smokers. Hemoglobin A1c are more elevated in smokers while glycemia are more elevated in non-smokers. We rapport some reason over these divergent results. Hemoglobin A1c elevated in smokers suggest that the tobacco has negative influence on glucose metabolism.
烟草促发动脉粥样硬化的机制尚不清楚。吸烟者的高血糖可能是一个促成因素。我们对一个由1379名平均年龄38岁的男性煤矿工人组成的工作人群中烟草、空腹血糖和糖化血红蛋白之间的数值关系进行了研究。我们发现吸烟者和非吸烟者之间糖化血红蛋白和空腹血糖值存在显著差异。吸烟者的糖化血红蛋白升高更明显，而非吸烟者的血糖升高更明显。我们阐述了导致这些不同结果的一些原因。吸烟者糖化血红蛋白升高表明烟草对葡萄糖代谢有负面影响。
