Beck C, Benning C D, Stange G
Acta Otolaryngol. 1975 Mar-Apr;79(3-4):297-303.
62 guinea-pigs were treated twice exposed to white noise, 105 dB SPL, 20 to 20 000 cps for 40 minutes with a break of 24 hours in between. The cochlear microphonics (CM), the compound action potentials of the acoustic nerve (NAP) and the slow evoked potentials (SEP) were recorded. There were different degrees of damage to be seen in the acoustic biopotentials. The CM showed little damages only whereas the compound action potentials of the hearing nerve showed significant impairments in excitation and in adaptation. In the SEP excitation was decreased. From this, we may conclude that the CM must not be the only criterion for judging damages in the acoustic system after acoustic trauma. Furthermore we see that the central acoustic pathway is capable to compensate peripheric hearing damages within a short time. An additional exposure to noise does not necessarily add or cumulate the degrees of damage.
62只豚鼠接受两次白噪声暴露，强度为105 dB SPL，频率范围20至20000 cps，每次40分钟，中间间隔24小时。记录了耳蜗微音电位（CM）、听神经复合动作电位（NAP）和慢诱发电位（SEP）。在听觉生物电位中可见不同程度的损伤。CM仅显示轻微损伤，而听神经复合动作电位在兴奋和适应方面显示出明显损害。SEP的兴奋度降低。由此我们可以得出结论，CM一定不是判断声创伤后听觉系统损伤的唯一标准。此外，我们发现中枢听觉通路能够在短时间内补偿外周听力损伤。再次暴露于噪声不一定会增加或累积损伤程度。
