Bjørnland T, Refsum S B
Department of Oral Surgery and Oral Medicine, Faculty of Dentistry, University of Oslo, Norway.
Oral Surg Oral Med Oral Pathol. 1994 Jun;77(6):572-8. doi: 10.1016/0030-4220(94)90313-1.
Histopathologic examination was performed of the disk and the posterior attachment extirpated from 17 temporomandibular joints from 15 patients with chronic arthritic disease. Seven patients had rheumatoid arthritis (including two with juvenile type), five had ankylosing spondylitis, and three had psoriatic arthropathy, which affected more joints than the temporomandibular joint. Specimens removed from 16 temporomandibular joints from 15 patients with internal derangement were used for histopathologic comparison. In both groups of patients, inflammatory changes were observed, but no specific histopathologic signs could distinguish the groups. Patients with chronic arthritic disease seemed to have more pronounced changes of vascular proliferation, perivascular cellular infiltrate, inflammatory cells, and fibrosis throughout the soft tissues. Destruction of the disk was another finding evident in patients with chronic arthritic temporomandibular joint disease; there was no visible disk structure in 8 of these 17 joints, compared with 1 of the 16 joints in the internal derangement group.
对15例患有慢性关节炎疾病患者的17个颞下颌关节切除的关节盘及后方附着组织进行了组织病理学检查。7例患者患有类风湿性关节炎（包括2例青少年型），5例患有强直性脊柱炎，3例患有银屑病关节炎（该疾病累及的关节比颞下颌关节更多）。取自15例患有颞下颌关节内紊乱患者的16个颞下颌关节的标本用于组织病理学比较。在两组患者中均观察到了炎症变化，但没有特定的组织病理学体征能够区分这两组。患有慢性关节炎疾病的患者在整个软组织中似乎有更明显的血管增生、血管周围细胞浸润、炎性细胞及纤维化改变。关节盘破坏是慢性关节炎性颞下颌关节疾病患者的另一个明显表现；在这17个关节中有8个没有可见的关节盘结构，而颞下颌关节内紊乱组的16个关节中有1个没有可见的关节盘结构。
