文献检索，用中文搜 PubMed

Suppr 超能文献

核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Gill T S, Leitner G, Porta S, Epple A

Department of Anatomy, Thomas Jefferson University, Philadelphia, PA 19107.

Comp Biochem Physiol C Comp Pharmacol Toxicol. 1993 Mar;104(3):489-95. doi: 10.1016/0742-8413(93)90023-e.

Long-term exposure (16 weeks) of the American eel, Anguilla rostrata, to an environmentally realistic concentration of cadmium (150 micrograms/l) causes a chronic elevation of plasma cortisol. During this time span, the eels retain the ability to respond to a weak additional stress (CO2 bubbles for 1 min) with a further increase in plasma cortisol. 2. Plasma glucose levels are significantly lowered after two weeks of exposure to cadmium (75 and 150 micrograms/l). However, subsequently they return close to control levels. 3. It is concluded that (a) a moderate cadmium pollution of the eel's habitat suffices to cause chronic stimulation of the eel's adrenocortical axis, and (b) the resulting continued hypercortisolemia must seriously affect the eel's metabolism, immunosystem, gonadal maturation, and ability to migrate to its spawning grounds.

将美洲鳗鲡（Anguilla rostrata）长期（16周）暴露于环境现实浓度的镉（150微克/升）中会导致血浆皮质醇长期升高。在此时间段内，鳗鱼保留了对微弱额外应激（CO2气泡1分钟）作出反应的能力，血浆皮质醇会进一步升高。2. 暴露于镉（75和150微克/升）两周后，血浆葡萄糖水平显著降低。然而，随后它们会恢复到接近对照水平。3. 得出的结论是：（a）鳗鱼栖息地的中度镉污染足以导致鳗鱼肾上腺皮质轴的慢性刺激，（b）由此产生的持续高皮质醇血症必然会严重影响鳗鱼的新陈代谢、免疫系统、性腺成熟以及洄游到产卵地的能力。

Gill T S, Leitner G, Porta S, Epple A

Department of Anatomy, Thomas Jefferson University, Philadelphia, PA 19107.

Comp Biochem Physiol C Comp Pharmacol Toxicol. 1993 Mar;104(3):489-95. doi: 10.1016/0742-8413(93)90023-e.

Long-term exposure (16 weeks) of the American eel, Anguilla rostrata, to an environmentally realistic concentration of cadmium (150 micrograms/l) causes a chronic elevation of plasma cortisol. During this time span, the eels retain the ability to respond to a weak additional stress (CO2 bubbles for 1 min) with a further increase in plasma cortisol. 2. Plasma glucose levels are significantly lowered after two weeks of exposure to cadmium (75 and 150 micrograms/l). However, subsequently they return close to control levels. 3. It is concluded that (a) a moderate cadmium pollution of the eel's habitat suffices to cause chronic stimulation of the eel's adrenocortical axis, and (b) the resulting continued hypercortisolemia must seriously affect the eel's metabolism, immunosystem, gonadal maturation, and ability to migrate to its spawning grounds.

将美洲鳗鲡（Anguilla rostrata）长期（16周）暴露于环境现实浓度的镉（150微克/升）中会导致血浆皮质醇长期升高。在此时间段内，鳗鱼保留了对微弱额外应激（CO2气泡1分钟）作出反应的能力，血浆皮质醇会进一步升高。2. 暴露于镉（75和150微克/升）两周后，血浆葡萄糖水平显著降低。然而，随后它们会恢复到接近对照水平。3. 得出的结论是：（a）鳗鱼栖息地的中度镉污染足以导致鳗鱼肾上腺皮质轴的慢性刺激，（b）由此产生的持续高皮质醇血症必然会严重影响鳗鱼的新陈代谢、免疫系统、性腺成熟以及洄游到产卵地的能力。