Pal'tsev M A, Zaĭrat'iants O V, Vetshev P S, Tuntsova O I
Arkh Patol. 1993;55(6):7-13.
Autoimmune thyroiditis (AT) are a heterogenous group of organ specific autoimmune diseases (AID) of the thyroid. Their pathogenesis is associated with predisposing, initiating and promoting factors. Morphogenesis is based on the hypersensitivity reaction of the immediate and delayed types with the development of acute or chronic immune inflammation in combination with atrophic and hyperplastic alterations of the parenchyma (reaction of neutralization and inactivation). AT can be subdivided into primary ones (Hashimoto's thyroiditis, chronic lymphocytic thyroiditis, chronic atrophic thyroiditis) and the secondary ones which are the manifestation of the organ-specific AID.
自身免疫性甲状腺炎（AT）是一组异质性的甲状腺器官特异性自身免疫性疾病（AID）。其发病机制与易感因素、起始因素和促进因素有关。形态发生基于速发型和迟发型超敏反应，伴有急性或慢性免疫炎症的发展，并伴有实质的萎缩和增生性改变（中和与失活反应）。AT可分为原发性（桥本甲状腺炎、慢性淋巴细胞性甲状腺炎、慢性萎缩性甲状腺炎）和继发性，继发性是器官特异性AID的表现。
