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[室间隔交通伴收缩期闭塞]

[Interventricular communication with systolic occlusion].

作者信息

Caffarena R, Bouchard F

出版信息

Arch Mal Coeur Vaiss. 1975 Apr;68(4):369-79.

PMID:816292
Abstract

Besides the ventricular defects of the "muscular" type with normal pressure values and which close during systole, there are ventricular septal defects in which systolic occlusion might be explained by other mechanisms; there are accompanied either by a stenosis of the outflow tract of the right ventricle (14 cases) or pulmonary hypertension (1 case). The diagnostic data were described, based mainly on the haemodynamic and angiocardiographic study. The hypotheses concerning the mechanism of occlusion in these cases were verified either on post-mortem or operative examinations.

摘要

除了“肌性”类型的室间隔缺损,其压力值正常且在收缩期闭合外,还有一些室间隔缺损,其收缩期闭塞可能由其他机制解释;这些缺损要么伴有右心室流出道狭窄(14例),要么伴有肺动脉高压(1例)。主要基于血流动力学和心血管造影研究描述了诊断数据。关于这些病例中闭塞机制的假设在尸检或手术检查中得到了验证。

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