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在体内给大鼠滴注石棉后,大鼠肺内会形成羟基自由基。

Hydroxyl radicals are formed in the rat lung after asbestos instillation in vivo.

作者信息

Schapira R M, Ghio A J, Effros R M, Morrisey J, Dawson C A, Hacker A D

机构信息

Division of Pulmonary and Critical Care Medicine, Medical College of Wisconsin, Milwaukee.

出版信息

Am J Respir Cell Mol Biol. 1994 May;10(5):573-9. doi: 10.1165/ajrcmb.10.5.8179922.

DOI:10.1165/ajrcmb.10.5.8179922
PMID:8179922
Abstract

The hydroxyl radical (.OH) has been implicated as a cause of lung injury following asbestos exposure. However, despite in vitro evidence associating asbestos with .OH production, there has been no demonstration of such generation in vivo. We tested the hypothesis that .OH is formed in the lungs of rats exposed to asbestos in vivo by using salicylate as a free radical trap. Asbestos was instilled intratracheally, and control rats were sham-exposed. Six to seven days after exposure, the rats were given salicylate, the lungs were isolated, and salicylate hydroxylation products (2,3- and 2,5-dihydroxybenzoic acid), reflecting .OH production, were measured. There was significantly more 2,3-dihydroxybenzoic acid in asbestos-exposed lungs compared with control lungs (2.32 +/- 0.360 nmol/lung versus 0.292 +/- 0.125, respectively, P < 0.001) and 2,5-dihydroxybenzoic acid (9.69 +/- 1.65 nmol/lung versus 2.63 +/- 0.274, respectively, P < 0.001). To demonstrate that the dihydroxybenzoic acid was actually formed in the lungs, the lungs from asbestos-exposed and control rats were isolated and perfused with either salicylate or 2,3-dihydroxybenzoic acid. In the lungs perfused with salicylate, 2,3- and 2,5-dihydroxybenzoic acids were detectable only in asbestos-exposed lungs. In the isolated lungs perfused with 2,3-dihydroxybenzoic acid, there was no significant difference in 2,3-dihydroxybenzoic acid between asbestos-exposed and control lungs. We conclude that asbestos stimulates .OH production in lungs in vivo.

摘要

羟基自由基(·OH)被认为是石棉暴露后导致肺损伤的一个原因。然而,尽管有体外证据表明石棉与·OH的产生有关,但尚未在体内证实这种产生。我们通过使用水杨酸盐作为自由基捕获剂,测试了在体内暴露于石棉的大鼠肺中形成·OH的假设。将石棉经气管内注入,对照组大鼠进行假暴露。暴露后6至7天,给大鼠注射水杨酸盐,分离肺组织,并测量反映·OH产生的水杨酸盐羟基化产物(2,3 - 和2,5 - 二羟基苯甲酸)。与对照肺相比,暴露于石棉的肺中2,3 - 二羟基苯甲酸明显更多(分别为2.32±0.360 nmol/肺和0.292±0.125,P < 0.001)以及2,5 - 二羟基苯甲酸(分别为9.69±1.65 nmol/肺和2.63±0.274,P < 0.001)。为了证明二羟基苯甲酸实际上是在肺中形成的,分离暴露于石棉和对照大鼠的肺,并用水杨酸盐或2,3 - 二羟基苯甲酸进行灌注。在用水杨酸盐灌注的肺中,仅在暴露于石棉的肺中可检测到2,3 - 和2,5 - 二羟基苯甲酸。在用2,3 - 二羟基苯甲酸灌注的分离肺中,暴露于石棉的肺和对照肺之间的2,3 - 二羟基苯甲酸没有显著差异。我们得出结论,石棉在体内刺激肺中·OH的产生。

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