Guérot C, Valère P E, Castillo-Fenoy A, Mayault C, Tricot R
Arch Mal Coeur Vaiss. 1976 Jul;69(7):671-80.
Nodal conduction has been studied in 44 normal subjects, 21 cases of 1st or 2nd degree AV block, 16 cases of LGL syndrome, and 19 patients with a distal infra-His AV block, by means of stimulation which was increased in regular steps of 5 beats per minute, and of extra-systolic stimulation. These two techniques demonstrate that the nodal response to an extrasystole or a regular tachycardia is not the same, and that different mechanisms are probably at play. With regular stimulation, the normal limits of 1/1 conduction lie between 140 and 200 per minute. A nodal block for a rate less than 130 per minute can be taken as indicative of a nodal defect. Delayed AH conduction is a much less precise criterion. It may reach a high level in normal subjects, and it is not certain that the finding of a PR interval of more than 200 msec. (AH of more than 140 msec.) at a normal rate is neccessarily indicative of nodal pathology. Cases of LGL syndrome are associated with short-circuits which are usually partial. The diverse responses observed in these different groups can be explained by modifications of structure within the A/V node itself.
通过以每分钟5次心跳的规律递增刺激以及期外收缩刺激,对44名正常受试者、21例一度或二度房室传导阻滞患者、16例LGL综合征患者和19例希氏束以下远端房室传导阻滞患者的结传导进行了研究。这两种技术表明,结对于期外收缩或规则性心动过速的反应并不相同,可能存在不同的机制。在规则刺激下,1:1传导的正常范围在每分钟140至200次之间。每分钟心率低于130次时出现的结性阻滞可被视为结性缺陷的指标。AH传导延迟是一个不太精确的标准。在正常受试者中它可能达到较高水平,并且在正常心率下PR间期超过200毫秒(AH超过140毫秒)这一发现不一定表明存在结性病变。LGL综合征病例与通常为部分性的短路有关。在这些不同组中观察到的不同反应可以通过房室结本身结构的改变来解释。