Moue Y, Kamio K, Tanigaki T, Hayashi Y, Kuwahira I, Takasaki Y, Ohta Y, Yamabayashi H
Department of Internal Medicine, Tokai University School of Medicine, Kanagawa, Japan.
Nihon Kyobu Shikkan Gakkai Zasshi. 1993 Aug;31(8):990-3.
We report a 71-year-old female patient with primary alveolar hypoventilation syndrome who received diaphragm pacing (DP) and developed obstructive sleep apnea syndrome (OSAS). Application of nCPAP markedly improved her nocturnal hypoxemia. The monitored polygrams before and after the application strongly suggested that the main mechanism of OSAS was an imbalance of activity between upper airway dilator muscles and pump muscles. Moreover, paradoxical movement of the rib cage is not necessarily due to upper airway obstruction. Monitoring of tidal volume and arterial oxygen saturation is essential for the diagnosis of DP-induced OSAS.
我们报告了一名71岁的患有原发性肺泡低通气综合征的女性患者，她接受了膈肌起搏（DP）并发展为阻塞性睡眠呼吸暂停综合征（OSAS）。应用无创持续气道正压通气（nCPAP）显著改善了她的夜间低氧血症。应用前后监测的多导睡眠图强烈提示，OSAS的主要机制是上气道扩张肌和呼吸泵肌之间活动的不平衡。此外，胸廓的矛盾运动不一定是由于上气道阻塞。监测潮气量和动脉血氧饱和度对于诊断DP诱发的OSAS至关重要。
