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[脑出血前的短暂性神经功能缺损。病理生理假说]

[Transient neurologic deficit preceding intracerebral hemorrhage. Physiopathological hypotheses].

作者信息

Gras P, Grosmaire N, Fayolle H, Vion P, Giroud M, Dumas R

机构信息

Service de Neurologie, Hôpital Général, Dijon.

出版信息

Rev Neurol (Paris). 1993;149(3):224-6.

PMID:8235219
Abstract

Seventeen out of 238 cases (7%) of intracerebral hemorrhage were preceded by transient neurological deficit. In two cases, intracerebral hemorrhage and transient neurological deficit were thought to be due to amyloid angiopathy, this diagnosis was confirmed at autopsy in one case. In one case, intracerebral hemorrhage and transient neurological deficit were due to a cavernoma. In the other 14 cases, hypertension was the major risk factor. In 3 of them, CT-scans made after the transient neurological deficit but before the intracerebral hemorrhage showed deep small hypodensities suggestive of lacunas. The mechanisms of transient neurological deficit preceding intracerebral hemorrhage are reviewed.

摘要

238例脑出血病例中有17例(7%)在出血前出现短暂性神经功能缺损。其中2例脑出血和短暂性神经功能缺损被认为是由淀粉样血管病引起的,1例经尸检确诊。1例脑出血和短暂性神经功能缺损是由海绵状血管瘤引起的。其他14例中,高血压是主要危险因素。其中3例在短暂性神经功能缺损后、脑出血前进行的CT扫描显示深部小低密度灶,提示腔隙性梗死。本文对脑出血前短暂性神经功能缺损的机制进行了综述。

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