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转基因高血压大鼠TGR(mREN2)27中小鼠Ren-2d肾素基因的个体发生调控

Ontogenetic regulation of mouse Ren-2d renin gene in transgenic hypertensive rats, TGR(mREN2)27.

作者信息

Zhao Y, Bader M, Kreutz R, Fernandez-Alfonso M, Zimmermann F, Ganten U, Metzger R, Ganten D, Mullins J J, Peters J

机构信息

German Institute for High Blood Pressure Research, University of Heidelberg.

出版信息

Am J Physiol. 1993 Nov;265(5 Pt 1):E699-707. doi: 10.1152/ajpendo.1993.265.5.E699.

Abstract

TGR(mREN2)27 is a new monogenetic rat model with fulminant hypertension, low kidney renin, and high extrarenal renin gene expression. This study characterizes and compares expression of the Ren-2 gene in TGR(mREN2)27 with that in DBA/2 mice and with renin gene expression in rats. Except in the submandibular gland, the tissue-specific expression of Ren-2 is similar in TGR(mREN2)27 and DBA/2. This demonstrates maintenance of tissue specificity. Organs that are involved in cardiovascular regulation, such as the adrenal gland, kidney, and brain, express the Ren-2 gene before hypertension has developed, consistent with the possibility of a causal relationship between transgene expression in these tissues and hypertension. Because these tissues express the renin gene in nontransgenic rats as well, we suggest that this model can be used to study the regulation of renin gene expression and its role in hypertension at these sites. In addition, as an indication that interactions may exist between blood pressure and renin gene expression, we describe reciprocal changes in blood pressure and Ren-2 mRNA levels in the kidney and brain.

摘要

TGR(mREN2)27是一种新的单基因大鼠模型,具有暴发性高血压、低肾素水平和高肾外肾素基因表达。本研究对TGR(mREN2)27中Ren-2基因的表达进行了表征,并与DBA/2小鼠中的表达以及大鼠中的肾素基因表达进行了比较。除了下颌下腺外,TGR(mREN2)27和DBA/2中Ren-2的组织特异性表达相似。这证明了组织特异性的维持。参与心血管调节的器官,如肾上腺、肾脏和大脑,在高血压发展之前就表达Ren-2基因,这与这些组织中的转基因表达与高血压之间存在因果关系的可能性一致。由于这些组织在非转基因大鼠中也表达肾素基因,我们认为该模型可用于研究肾素基因表达的调节及其在这些部位高血压中的作用。此外,作为血压与肾素基因表达之间可能存在相互作用的一个迹象,我们描述了肾脏和大脑中血压和Ren-2 mRNA水平的相互变化。

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