Scherlag B J, Wang X, Nakagawa H, Hirao K, Santoro I, Dugger D, Gwin R M, Peters L, Lazzara R, Jackman W M
University of Oklahoma Health Sciences Center, Oklahoma City.
J Am Vet Med Assoc. 1993 Oct 15;203(8):1147-52.
A new technique for the treatment of certain types of cardiac arrhythmias was used in a 3-year-old dog that was evaluated for incessant supraventricular tachycardia (220 to 280 beats/min), which had been refractory to several treatment regimens. The mechanism of supraventricular tachycardia was atrioventricular (AV) reentry, using a dorsoseptal accessory pathway (AP) for retrograde ventriculoatrial conduction (concealed AP). With the dog under general anesthesia and with fluoroscopic monitoring, electrode catheters were introduced into the heart via peripheral vessels. Electrical recordings allowed localization of the accessory AV pathway. Programmed electrical stimulation was used to verify the function of the abnormal AV connection. At the atrial insertion site of the AP, 2 applications of radiofrequency current (45 V, 21.6 W) were delivered to the dorsoseptal right atrium (near the coronary sinus ostium), which eliminated AP conduction and AV reentrant tachycardia. The dog has remained free of tachycardia and has not required medication during more than 1 year of follow-up.
一种治疗某些类型心律失常的新技术应用于一只3岁的犬,该犬因持续性室上性心动过速(220至280次/分钟)接受评估,该心律失常对多种治疗方案均无效。室上性心动过速的机制为房室(AV)折返,利用后间隔旁路(AP)进行逆向室房传导(隐匿性AP)。在犬全身麻醉并进行荧光透视监测的情况下,通过外周血管将电极导管插入心脏。电记录可定位房室旁路。程序电刺激用于验证异常房室连接的功能。在AP的心房插入部位,对右后间隔心房(靠近冠状窦口)进行了2次射频电流(45V,21.6W)施加,消除了AP传导和房室折返性心动过速。在超过1年的随访期间,该犬一直未出现心动过速,也未需要药物治疗。