Yamada Takumi, Huizar Jose F, McElderry Hugh T, Kay G Neal
Division of Cardiovascular Diseases, Cardiac Rhythm Management Laboratory, University of Alabama at Birmingham, VH B147, 1670 University Boulevard, 1530 3rd AVE S, Birmingham, AL 35294-0019, USA.
Europace. 2007 May;9(5):299-301. doi: 10.1093/europace/eum037. Epub 2007 Mar 15.
A 68-year-old woman with palpitations underwent electrophysiologic testing. During burst atrial pacing the PR interval exceeded the RR interval and induced a supraventricular tachycardia consistent with a typical AV nodal reentrant tachycardia (AVNRT). Radiofrequency ablation of the slow pathway during the tachycardia immediately produced 2 : 1 AV conduction. After slow AV nodal pathway ablation an atrial tachycardia (AT) remained inducible with the earliest atrial activation around the HB region. Radiofrequency ablation at the site of earliest atrial activation interrupted the AT without AV block. AT originating from the HB region with slow pathway conduction may mimic typical AVNRT.
一名68岁心悸女性接受了电生理检查。在短阵房性起搏期间,PR间期超过RR间期,并诱发了与典型房室结折返性心动过速(AVNRT)一致的室上性心动过速。心动过速期间对慢径路进行射频消融立即产生了2:1房室传导。慢径路消融后,仍可诱发起源于希氏束(HB)区域附近最早心房激动的房性心动过速(AT)。在最早心房激动部位进行射频消融中断了AT,未出现房室传导阻滞。起源于HB区域且伴有慢径路传导的AT可能会酷似典型AVNRT。