Gibbels E, Weinmann S, Schorre W
MMW Munch Med Wochenschr. 1976 Dec 17;118(51):1657-60.
By investigation of the carbohydrate metabolism in 357 patients with polyneuropathy observed in hospital in recent years, and by statistical comparison with a control group in the literature, an attempt was made to find out whether subclinical diabetes can cause diabetic polyneuropathy to a greater extent. In spite of the more frequent occurrence in the total number of patients, a more significant etiological role for this type of metabolic disorder in polyneuropathy cannot be concluded from the results. Whether a more unspecific, general polyneuropathy-promoting influence is to be ascribed to it must remain open.
通过对近年来住院观察的357例多发性神经病患者的碳水化合物代谢进行调查，并与文献中的对照组进行统计学比较，试图找出亚临床糖尿病是否在更大程度上导致糖尿病性多发性神经病。尽管在患者总数中这种情况更频繁出现，但从结果中不能得出这种代谢紊乱在多发性神经病中具有更显著病因学作用的结论。是否应将更非特异性的、促进一般性多发性神经病的影响归因于它仍有待确定。
