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Acute promyelocytic leukemia: from genetics to treatment.

作者信息

Grignani F, Fagioli M, Alcalay M, Longo L, Pandolfi P P, Donti E, Biondi A, Lo Coco F, Grignani F, Pelicci P G

机构信息

Istituto di Clinica Medica I, University of Perugia, Policlinico Monteluce, Italy.

出版信息

Blood. 1994 Jan 1;83(1):10-25.

PMID:8274729
Abstract
摘要

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Acute promyelocytic leukemia: from genetics to treatment.急性早幼粒细胞白血病:从遗传学到治疗
Blood. 1994 Jan 1;83(1):10-25.
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[Role of retinoic acid in the treatment of acute promyelocytic leukemia].[维甲酸在急性早幼粒细胞白血病治疗中的作用]
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Retinoic acid and arsenic: towards oncogene-targeted treatments of acute promyelocytic leukaemia.维甲酸与砷:迈向急性早幼粒细胞白血病的癌基因靶向治疗
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Retinoic acid regulatory pathways, chromosomal translocations, and acute promyelocytic leukemia.维甲酸调节通路、染色体易位与急性早幼粒细胞白血病
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[All-trans retinoic acid therapy in acute promyelocytic leukemia--current status and prospect].[全反式维甲酸治疗急性早幼粒细胞白血病——现状与展望]
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Pathogenetic role of the PML1RAR alpha fusion protein in acute promyelocytic leukemia.PML1RARα融合蛋白在急性早幼粒细胞白血病中的致病作用。
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[Arsenic and retinoic acid, toward targeted treatments of acute promyelocytic anemia?].[砷与维甲酸,走向急性早幼粒细胞白血病的靶向治疗?]
C R Seances Soc Biol Fil. 1998;192(2):273-81.
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The PML-RAR alpha gene product of the t(15;17) translocation inhibits retinoic acid-induced granulocytic differentiation and mediated transactivation in human myeloid cells.t(15;17)易位产生的PML-RARα基因产物可抑制人髓系细胞中视黄酸诱导的粒细胞分化并介导反式激活。
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