Höhmann D, Lohmann T, Schwager K
Universitäts-Hals-Nasen-Ohrenklinik Würzburg.
Laryngorhinootologie. 1993 May;72(5):231-5. doi: 10.1055/s-2007-997890.
In 50 patients auditory threshold and brain stem evoked potential studies were carried out before and after myelography. Due to the analysis of amplitudes and latencies of auditory brain stem measurements, significant functional disorders of the hearing organ and the auditory pathway could be demonstrated. In most of the patients these functional disorders were found to be subclinical, whereas 12 patients showed alterations extending from a subjectively slight hearing loss to an audiometrically objectified acute hearing loss depending on its intensity in each case. The reasons of these functional disorders could not be clarified. An open cochlear aqueduct through which perilymph enters the subarachnoidal space leading to a secondary endolymphatic hydrops can be considered as the cause in cases where manifest symptoms develop. The changes in brain stem audiometry can be additionally explained by changes in osmolality of the inner ear fluids which may lead to the development of an endolymphatic hydrops.
对50例患者在脊髓造影前后进行了听阈和脑干诱发电位研究。通过分析听觉脑干测量的波幅和潜伏期,发现听觉器官和听觉通路存在明显的功能障碍。大多数患者的这些功能障碍为亚临床状态,而12例患者根据其各自的强度表现出从主观上轻微的听力损失到听力测定客观化的急性听力损失等不同程度的改变。这些功能障碍的原因尚不清楚。在出现明显症状的病例中,可认为是外淋巴通过开放的蜗水管进入蛛网膜下腔导致继发性内淋巴积水所致。内耳液渗透压的变化可能导致内淋巴积水,这也可以进一步解释脑干听力测定的变化。