Satake S, Heijma K, Sakamoto Y, Suzuki F, Sano T
J Electrocardiol. 1977 Jan;10(1):71-6. doi: 10.1016/s0022-0736(77)80035-6.
In a patient with documented paroxysmal junctional tachycardia (PJT) electrophysiologic studies were performed using an extrastimulus technique. At an A1-A2 interval of 360 msec, atrial extrastimulus revealed sudden prolongation of an A2-H2 interval from 370 to 540 msec and PJT ensued. This finding was consistent with antegrade dual A-V nodal pathways. On the other hand, at a V1-V2 interval of 540 msec, ventricular estrastimulus showed a jump in ventriculo-atrial (V-A) conduction time with evidence of delay in the A-V node from 285 to 565 msec and a ventricular echo followed. This finding was consistent with retrograde dual A-V nodal pathways. Mechanisms of bidirectional dual A-V nodal pathways are discussed.
在一名记录有阵发性交界性心动过速(PJT)的患者中,采用额外刺激技术进行了电生理研究。在A1 - A2间期为360毫秒时,心房额外刺激显示A2 - H2间期从370毫秒突然延长至540毫秒,随后出现PJT。这一发现与顺行性双房室结径路相符。另一方面,在V1 - V2间期为540毫秒时,心室额外刺激显示室房(V - A)传导时间跳跃,伴有房室结延迟,从285毫秒至565毫秒,随后出现心室回波。这一发现与逆行性双房室结径路相符。本文讨论了双向双房室结径路的机制。