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房室结间隙中房室传导延迟的机制。

The mechanism of A-V conduction delay in A-V nodal gap.

作者信息

Ohe T, Matsuhisa M, Kamakura S, Hirata Y, Ejiri N, Shimomura K

出版信息

J Electrocardiol. 1982 Oct;15(4):381-8. doi: 10.1016/s0022-0736(82)81011-x.

Abstract

Initial block in the His-Purkinje system was demonstrated in 17 of 71 patients during atrial extrastimulus testing. Of the 17 patients who demonstrated initial block in the His-Purkinje system, ten patients had A-V nodal gap (a type 1 gap). Of these ten patients, progressive increase in A-V nodal conduction was responsible for a resumption of A-V conduction in six patients, dual pathways within the A-V node was responsible in three patients, and the presence of an extra nodal pathway of the A-V node was responsible in one patient.

摘要

在71例患者的心房期外刺激测试中,有17例在希氏-浦肯野系统出现初始阻滞。在这17例希氏-浦肯野系统出现初始阻滞的患者中,10例存在房室结间隙(1型间隙)。在这10例患者中,6例患者房室结传导的逐渐增加导致房室传导恢复,3例患者是房室结内双径路导致,1例患者是房室结存在结外通路导致。

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