Maresh H, Klimek J J, Quintiliani R
Chest. 1977 Mar;71(3):410-3. doi: 10.1378/chest.71.3.410.
A patient with evidence of myocardial abnormalities and hemolytic anemia is described, in whom the responsible pathogen appeared to be Mycoplasma pneumoniae (as indicated by a 64-fold rise in complement-fixation titers, and by a change in cold-agglutinin titers from 1:8 to 1:4,096). Both cardiac and hematologic problems occurred during the recovery phase from pneumonia and were associated with marked deterioration in the patient's clinical status. Electrocardiographic and serum enzymatic changes mimicked the patterns seen in acute myocardial infarction.
本文描述了一名患有心肌异常和溶血性贫血的患者,其致病病原体似乎是肺炎支原体(补体结合试验滴度升高64倍,冷凝集素滴度从1:8变为1:4096表明了这一点)。心脏和血液学问题均出现在肺炎恢复期,且与患者临床状况的显著恶化相关。心电图和血清酶学变化类似急性心肌梗死所见的模式。