Matioli G T
USC, Los Angeles 90033.
Med Hypotheses. 1993 Jun;40(6):326-8. doi: 10.1016/0306-9877(93)90211-8.
The mechanism of SCE discussed in previous papers (3, 4) is adapted to account for the absence of kinks on chromatids experiencing an exchange limited to their KSs. The paper ends with a brief digression on kinks. Equivalent to solitons, such dynamic malformations can function as carriers of information along the chromatid stack.
先前论文（3, 4）中讨论的姐妹染色单体交换（SCE）机制，被用于解释在仅限于其KSs的交换中染色单体上没有纽结的现象。本文最后简要离题讨论了纽结。与孤子类似，这种动态畸形可作为沿染色单体堆栈的信息载体。
