Inhibitory action of Lambert-Eaton syndrome IgG on calcium currents in a thyroid C-cell line.
作者信息
Kim Y I, Blandino J K, O'Shaughnessy T J
机构信息
Department of Biomedical Engineering, University of Virginia Health Sciences Center, Charlottesville 22908.
出版信息
Ann N Y Acad Sci. 1993 Jun 21;681:398-401. doi: 10.1111/j.1749-6632.1993.tb22919.x.
Abstract
摘要
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Inhibitory action of Lambert-Eaton syndrome IgG on calcium currents in a thyroid C-cell line.兰伯特-伊顿综合征IgG对甲状腺C细胞系钙电流的抑制作用。
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Ca2+ channels as targets of neurological disease: Lambert-Eaton Syndrome and other Ca2+ channelopathies.作为神经疾病靶点的钙离子通道：兰伯特-伊顿综合征及其他钙离子通道病
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Passive transfer of Lambert-Eaton syndrome to mice induces dihydropyridine sensitivity of neuromuscular transmission.将兰伯特-伊顿综合征被动转移至小鼠会诱导神经肌肉传递的二氢吡啶敏感性。
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Lambert-Eaton sera reduce low-voltage and high-voltage activated Ca2+ currents in murine dorsal root ganglion neurons.兰伯特-伊顿血清可降低小鼠背根神经节神经元中低电压和高电压激活的Ca2+电流。
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