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Gangliosides as receptors for bacterial enterotoxins.

作者信息

Fishman P H, Pacuszka T, Orlandi P A

机构信息

Membrane Biochemistry Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892.

出版信息

Adv Lipid Res. 1993;25:165-87.

PMID:8396312
Abstract
摘要

相似文献

1
Gangliosides as receptors for bacterial enterotoxins.神经节苷脂作为细菌肠毒素的受体
Adv Lipid Res. 1993;25:165-87.
2
Cholera toxin and related enterotoxins as potent immune modulators.霍乱毒素及相关肠毒素作为强效免疫调节剂。
Symp Ser Soc Appl Microbiol. 1998;27:26S-34S. doi: 10.1046/j.1365-2672.1998.0840s126s.x.
3
Modulation of enterotoxin binding and function in vitro and in vivo.
J Infect Dis. 1988 Mar;157(3):557-64. doi: 10.1093/infdis/157.3.557.
4
Comparison of receptors for cholera and Escherichia coli enterotoxins in human intestine.人肠道中霍乱毒素和大肠杆菌肠毒素受体的比较。
Gastroenterology. 1985 Jul;89(1):27-35. doi: 10.1016/0016-5085(85)90741-3.
5
Receptor blocking, cholera, and Escherichia coli toxins.受体阻断、霍乱毒素和大肠杆菌毒素。
J Infect Dis. 1988 Nov;158(5):1147-8. doi: 10.1093/infdis/158.5.1147-a.
6
Characterization of the receptor for cholera toxin and Escherichia coli heat-labile toxin in rabbit intestinal brush borders.兔肠刷状缘中霍乱毒素和大肠杆菌不耐热毒素受体的特性研究
Biochem J. 1986 Sep 1;238(2):313-22. doi: 10.1042/bj2380313.
7
Binding of class II Escherichia coli enterotoxins to mouse Y1 and intestinal cells.II类大肠杆菌肠毒素与小鼠Y1细胞和肠道细胞的结合。
Infect Immun. 1992 Jul;60(7):2870-3. doi: 10.1128/iai.60.7.2870-2873.1992.
8
Receptors for cholera toxin and Escherichia coli heat-labile enterotoxin revisited.霍乱毒素和大肠杆菌不耐热肠毒素受体再探讨
Prog Brain Res. 1994;101:163-77. doi: 10.1016/s0079-6123(08)61947-0.
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Rabbit intestinal glycoprotein receptor for Escherichia coli heat-labile enterotoxin lacking affinity for cholera toxin.对霍乱毒素缺乏亲和力的兔源大肠杆菌热不稳定肠毒素肠道糖蛋白受体
Infect Immun. 1982 Nov;38(2):424-33. doi: 10.1128/iai.38.2.424-433.1982.
10
[Similarities and differences in the structure and mechanism of action of bacterial, secretogenic enterotoxins].[细菌分泌型肠毒素的结构与作用机制的异同]
Z Gastroenterol. 1986 Sep;24 Suppl 3:19-24.

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