Muirhead's syndrome and medullipin. A new syndrome and a unique hypotensive agent.
作者信息
Swales J D
出版信息
Br Heart J. 1993 Sep;70(3):211. doi: 10.1136/hrt.70.3.211.
Abstract
摘要
相似文献
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Br Heart J. 1993 Sep;70(3):211. doi: 10.1136/hrt.70.3.211.
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本文引用的文献
1
Lipomedullipinoma: a source of hypermedullipinemia.脂肪髓母细胞瘤：高髓磷脂血症的一个来源。
Blood Press. 1993 Sep;2(3):183-8. doi: 10.3109/08037059309077549.
2
Suppression of tonic sympathetic nerve activity by depressor agents released from the declipped kidney.去夹肾释放的降压物质对交感神经紧张性活动的抑制作用
Acta Physiol Scand. 1982 Sep;116(1):93-5. doi: 10.1111/j.1748-1716.1982.tb10603.x.
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Antihypertensive functions of the kidney: Arthur C. Corcoran memorial lecture.肾脏的降压功能：亚瑟·C·科科伦纪念讲座
Hypertension. 1980 Jul-Aug;2(4):444-64. doi: 10.1161/01.hyp.2.4.444.
4
Hypertension produced by chemical renal medullectomy: evidence for a renomedullary vasodepressor function in the rat.化学性肾髓质切除所致高血压：大鼠肾髓质血管舒张降压功能的证据
Clin Sci (Lond). 1984 Nov;67(5):521-8. doi: 10.1042/cs0670521.
5
Production of renomedullary prostaglandins by renomedullary interstitial cells grown in tissue culture.组织培养中生长的肾髓质间质细胞产生肾髓质前列腺素。
Circ Res. 1972 Sep;31(9):Suppl 2:161-72.
6
Reversal of renovascular hypertension: role of the renal medulla.
Can J Physiol Pharmacol. 1987 Aug;65(8):1566-71. doi: 10.1139/y87-246.
7
Renal vasodepressor mechanism: characterization by chemical medullectomy.肾血管减压机制：通过化学性肾髓质切除进行特征描述。
Am J Med Sci. 1988 Apr;295(4):241-5. doi: 10.1097/00000441-198804000-00004.
8
The renal antihypertensive endocrine function: its relation to cytochrome P-450.肾脏的降压内分泌功能：及其与细胞色素P-450的关系。
J Hypertens. 1989 May;7(5):361-9. doi: 10.1097/00004872-198905000-00003.
9
Do angiotensin converting enzyme inhibitors lower blood pressure in the rat partly via the humorally mediated antihypertensive system of the renal medulla?血管紧张素转换酶抑制剂是否部分通过肾髓质的体液介导性降压系统来降低大鼠的血压？
J Hypertens. 1990 Jun;8(6):501-13. doi: 10.1097/00004872-199006000-00002.
10
Persistent hypotension associated with hypermedullipinemia: a new syndrome.与高髓质素血症相关的持续性低血压：一种新综合征。
Blood Press. 1992 Oct;1(3):138-48. doi: 10.3109/08037059209077509.
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