慢性肠道炎症：细胞因子或T细胞受体突变小鼠中的意外结果。 - Suppr

Chronic intestinal inflammation: an unexpected outcome in cytokine or T cell receptor mutant mice.

作者信息

Strober W, Ehrhardt R O

机构信息

National Institutes of Health, Mucosal Immunity Section, National Institute of Allergy and Infectious Diseases, Bethesda, Maryland 20892-0010.

出版信息

Cell. 1993 Oct 22;75(2):203-5. doi: 10.1016/0092-8674(93)80062-j.

DOI:10.1016/0092-8674(93)80062-j

PMID:8402907

Abstract

摘要

相似文献

Chronic intestinal inflammation: an unexpected outcome in cytokine or T cell receptor mutant mice.慢性肠道炎症：细胞因子或T细胞受体突变小鼠中的意外结果。

Cell. 1993 Oct 22;75(2):203-5. doi: 10.1016/0092-8674(93)80062-j.

Spontaneous development of inflammatory bowel disease in T cell receptor mutant mice.T细胞受体突变小鼠中炎症性肠病的自发发展。

Cell. 1993 Oct 22;75(2):274-82. doi: 10.1016/0092-8674(93)80069-q.

Cytokine imbalance and autoantibody production in T cell receptor-alpha mutant mice with inflammatory bowel disease.炎症性肠病的T细胞受体α突变小鼠中的细胞因子失衡与自身抗体产生

J Exp Med. 1996 Mar 1;183(3):847-56. doi: 10.1084/jem.183.3.847.

Experimental models to study molecular mechanisms underlying intestinal inflammation.

Ann N Y Acad Sci. 1998 Nov 17;859:85-95. doi: 10.1111/j.1749-6632.1998.tb11113.x.

Insights from mouse models of colitis.结肠炎小鼠模型的见解。

J Leukoc Biol. 2000 Mar;67(3):267-78. doi: 10.1002/jlb.67.3.267.

Cytokine mRNA expression in intestinal tissue of interleukin-2 deficient mice with bowel inflammation.白细胞介素-2缺陷型肠炎小鼠肠道组织中细胞因子mRNA的表达

Gut. 1997 Dec;41(6):793-800. doi: 10.1136/gut.41.6.793.

Specific-pathogen-free conditions enhance inflammatory bowel disease in T-cell receptor knockout, but not C3H/HeJBir mice.无特定病原体条件会加重T细胞受体敲除小鼠而非C3H/HeJBir小鼠的炎症性肠病。

Lab Anim Sci. 1998 Oct;48(5):432-3.

Cells and cytokines in the pathogenesis of inflammatory bowel disease: new insights from mouse T cell transfer models.

Exp Clin Immunogenet. 2000;17(3):115-29. doi: 10.1159/000019131.

Lessons from genetic models of inflammatory bowel disease.炎症性肠病遗传模型的经验教训。

Acta Gastroenterol Belg. 1997 Apr-Jun;60(2):163-5.

Specific-pathogen-free conditions enhance inflammatory bowel disease in T-cell receptor knockout, but not C3H/HeJBir mice.

Lab Anim Sci. 1997 Dec;47(6):650-5.

引用本文的文献

Intestinal single-cell atlas reveals novel lymphocytes in pigs with similarities to human cells.肠道单细胞图谱揭示了与人类细胞具有相似性的新型猪淋巴细胞。

Life Sci Alliance. 2022 Aug 22;5(10). doi: 10.26508/lsa.202201442. Print 2022 Oct.

Loricrin: Past, Present, and Future.角蛋白 1: 过去、现在和未来。

Int J Mol Sci. 2020 Mar 25;21(7):2271. doi: 10.3390/ijms21072271.

IL-10-producing CD4(+) T cells negatively regulate fucosylation of epithelial cells in the gut.产生白细胞介素-10的CD4(+) T细胞对肠道上皮细胞的岩藻糖基化起负向调节作用。

Sci Rep. 2015 Nov 2;5:15918. doi: 10.1038/srep15918.

Oral administration of probiotic bacteria (E. coli Nissle, E. coli O83, Lactobacillus casei) influences the severity of dextran sodium sulfate-induced colitis in BALB/c mice.口服益生菌（大肠杆菌Nissle、大肠杆菌O83、干酪乳杆菌）会影响葡聚糖硫酸钠诱导的BALB/c小鼠结肠炎的严重程度。

Folia Microbiol (Praha). 2006;51(5):478-84. doi: 10.1007/BF02931595.

Induction of colitis by a CD4+ T cell clone specific for a bacterial epitope.由针对细菌表位的CD4 + T细胞克隆诱导结肠炎。

Proc Natl Acad Sci U S A. 2003 Dec 23;100(26):15830-5. doi: 10.1073/pnas.2534546100. Epub 2003 Dec 12.

Development of colitis in signal transducers and activators of transcription 6-deficient T-cell receptor alpha-deficient mice: a potential role of signal transducers and activators of transcription 6-independent interleukin-4 signaling for the generation of Th2-biased pathological CD4+ betabetaT cells.信号转导及转录激活因子6缺陷型T细胞受体α缺陷型小鼠中结肠炎的发生：信号转导及转录激活因子6非依赖性白细胞介素-4信号在Th2偏向性病理性CD4+ betabetaT细胞生成中的潜在作用。

Am J Pathol. 2003 Jan;162(1):263-71. doi: 10.1016/s0002-9440(10)63817-5.

Cloning and expression of a Helicobacter bilis immunoreactive protein.胆幽门螺杆菌免疫反应蛋白的克隆与表达

Clin Diagn Lab Immunol. 2002 May;9(3):627-32. doi: 10.1128/cdli.9.3.627-632.2002.

The role of microflora in the development of intestinal inflammation: acute and chronic colitis induced by dextran sulfate in germ-free and conventionally reared immunocompetent and immunodeficient mice.微生物群在肠道炎症发展中的作用：葡聚糖硫酸钠在无菌及常规饲养的免疫健全和免疫缺陷小鼠中诱导的急性和慢性结肠炎。

Folia Microbiol (Praha). 2001;46(6):565-72. doi: 10.1007/BF02818004.

Development of antigen induced colitis in SCID mice reconstituted with spleen derived memory type CD4(+) CD45RB(+) T cells.用脾脏来源的记忆型CD4(+)CD45RB(+)T细胞重建的SCID小鼠中抗原诱导性结肠炎的发展

Gut. 2002 Mar;50(3):299-306. doi: 10.1136/gut.50.3.299.

Helicobacter hepaticus-induced colitis in interleukin-10-deficient mice: cytokine requirements for the induction and maintenance of intestinal inflammation.肝螺杆菌诱导白细胞介素-10缺陷小鼠发生结肠炎：肠道炎症诱导和维持所需的细胞因子

Infect Immun. 2001 Jul;69(7):4232-41. doi: 10.1128/IAI.69.7.4232-4241.2001.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验

Chronic intestinal inflammation: an unexpected outcome in cytokine or T cell receptor mutant mice.

作者信息

机构信息

出版信息

相似文献

引用本文的文献

文献AI研究员

用中文搜PubMed

文档翻译

Suppr 超能文献

相似文献

引用本文的文献