Machemer R
Department of Ophthalmology, Duke University School of Medicine, Durham, NC 27710.
Graefes Arch Clin Exp Ophthalmol. 1993 Jul;231(7):389-94. doi: 10.1007/BF00919646.
Five cases of traction retinal detachment occurring later in life as a sequel of cicatricial retinopathy of prematurity or showing the clinical picture of retinopathy of prematurity are reported. They presented with taut membranes in vitreous cavity, causing traction retinal detachment, and often showed preretinal membranes. These membranes were collagen-rich and contained cells with glial characteristics. They seemed to be continuously produced on the surface of the retina from which they detached sometimes in multiple generations. It is likely that chronic exudation from vascular abnormalities is a stimulus for this proliferation. These cases are very similar to other vitreoretinal proliferations in association with vascular abnormalities (Coats' and von Hippel disease, exudative vitreoretinopathy).
本文报告了5例迟发性牵引性视网膜脱离病例，这些病例均为早产儿瘢痕性视网膜病变的后遗症，或表现出早产儿视网膜病变的临床症状。这些病例的玻璃体腔内可见紧张的膜，导致牵引性视网膜脱离，且常伴有视网膜前膜。这些膜富含胶原蛋白，含有具有神经胶质特征的细胞。它们似乎在视网膜表面持续产生，有时会多次从视网膜表面脱离。血管异常引起的慢性渗出可能是这种增殖的刺激因素。这些病例与其他伴有血管异常的玻璃体视网膜增殖性病变（科茨病、冯·希佩尔病、渗出性玻璃体视网膜病变）非常相似。
