Bhatia S, Sharma B S, Mathuriya S N, Pathak A, Khosla V K
Department of Neurosurgery, Postgraduate Institute of Medical Education and Research, Chandigarh, India.
Paraplegia. 1993 Aug;31(8):542-4. doi: 10.1038/sc.1993.88.
A patient with a complete fracture dislocation of the C6 vertebra without any neurological deficit is presented. The absence of neurological changes in an area of the spinal canal which is almost completely filled with the spinal cord is remarkable. The mechanism of injury in this patient is believed to be hyperextension associated with significant axial loading. Hyperextension first ruptured the anterior and the posterior longitudinal ligaments and then injured the pedicles of C6. The associated axial loading resulted in a 'bursting' of the posterior elements, thereby providing room for the spinal cord to move dorsally with the avoidance of a major neurological deficit.
本文报告了一名C6椎体完全骨折脱位但无任何神经功能缺损的患者。在几乎完全被脊髓占据的椎管区域未出现神经功能改变，这一点值得注意。该患者的损伤机制被认为是与显著轴向负荷相关的过伸。过伸首先导致前纵韧带和后纵韧带断裂，随后损伤C6椎弓根。相关的轴向负荷导致后部结构“爆裂”，从而为脊髓向后移动提供了空间，避免了严重的神经功能缺损。
