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暴露于低pH值对细胞表面表达的流感血凝素侧向流动性的影响:流动性抑制与失活之间的相关性

Effects of exposure to low pH on the lateral mobility of influenza hemagglutinin expressed at the cell surface: correlation between mobility inhibition and inactivation.

作者信息

Gutman O, Danieli T, White J M, Henis Y I

机构信息

Department of Biochemistry, George S. Wise Faculty of Life Sciences, Tel Aviv University, Israel.

出版信息

Biochemistry. 1993 Jan 12;32(1):101-6. doi: 10.1021/bi00052a014.

DOI:10.1021/bi00052a014
PMID:8418830
Abstract

To investigate the possible role of viral glycoprotein mobility in membrane fusion, fluorescence photobleaching recovery was employed to study the effects of exposure to mildly acidic pH (required to convert many viral fusion proteins to the fusion-active form) on the lateral mobility of influenza hemagglutinin (HA) proteins expressed at the surface of transfected cells. HA proteins from two different strains were compared: X:31 HA, which is activated by a brief exposure to pH 4.9 but is irreversibly inactivated at longer exposure times, and HA from A/Japan/305/57, which is relatively stable to inactivation at this pH [Puri, A., Booy, F.P., Doms, R.W., White, J.M., & Blumenthal, R. (1990) J. Virol. 64, 3824-3832]. The HA proteins from both strains, expressed in CV-1 cells using VS-40 vectors, exhibited relatively unrestricted lateral diffusion at the cell surface. The high mobility persisted following a brief exposure (1 min) to pH 4.9 to mediate conversion to the fusogenic state. Longer times (up to 15 min) of preincubation at pH 4.9 inhibited the lateral mobility of X:31 HA (the lateral diffusion rate was markedly reduced, followed by immobilization) but not of A/Japan HA, whose fusion activity is resistant to such treatment. Inhibition of the lateral mobility of X:31 HA due to preincubation at low pH was not specific to the CV-1 cells and was found also in a CHO cell line stably expressing this protein. The results presented demonstrate a close correlation between loss of mobility and inactivation of fusogenic activity, in accord with the notion that lateral motion of the HA proteins is required for fusion.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

为了研究病毒糖蛋白流动性在膜融合中的可能作用,采用荧光光漂白恢复技术来研究暴露于轻度酸性pH值(将许多病毒融合蛋白转化为融合活性形式所必需的)对转染细胞表面表达的流感血凝素(HA)蛋白侧向流动性的影响。比较了来自两种不同毒株的HA蛋白:X:31 HA,短暂暴露于pH 4.9可被激活,但长时间暴露会不可逆地失活;以及来自A/日本/305/57的HA,在该pH值下对失活相对稳定[普里,A.,布伊,F.P.,多姆斯,R.W.,怀特,J.M.,& 布卢门撒尔,R.(1990年)《病毒学杂志》64卷,3824 - 3832页]。使用VS - 40载体在CV - 1细胞中表达的两种毒株的HA蛋白,在细胞表面均表现出相对不受限制的侧向扩散。短暂暴露(1分钟)于pH 4.9以介导转化为融合状态后,高流动性持续存在。在pH 4.9下预孵育较长时间(长达15分钟)会抑制X:31 HA的侧向流动性(侧向扩散速率显著降低,随后固定),但不会抑制A/日本HA的侧向流动性,其融合活性对这种处理具有抗性。由于在低pH下预孵育导致X:31 HA侧向流动性的抑制并非CV - 1细胞所特有,在稳定表达该蛋白的CHO细胞系中也有发现。所呈现的结果表明流动性丧失与融合活性失活之间存在密切相关性,这与HA蛋白的侧向运动是融合所必需的观点一致。(摘要截短于250字)

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