Stewart W K, Guirgis H A, Sanderson J, Taylor W
Br J Ind Med. 1977 Feb;34(1):26-31. doi: 10.1136/oem.34.1.26.
The use of mercuric chloride as an histological fixative was associated with high environmental atmospheric concentrations of mercury vapour (up to 0-5 nmol/l) as well as mercury compounds (total Hg to 1-0 nmol/l). Technicians exposed to this environment showed increased urinary mercury (median value 265 nmol/24h) and protein outputs (median value 117 mg protein/24h). Routine control measures, ventilation and careful handling of mercuric chloride solutions, reduced the level of atmospheric mercury vapour levels to within acceptable limits (threshold limit values 0-01 mg/m3 (0-05 nmol/l) alkul compounds and 0-05 mg/m3 (0-25 nmol/l) for all forms except alkyl). This reduction was associated with the disappearance of trace proteinuria from the technicians' urine. Contamination of histology laboratories by mercuric chloride should be minimised.
使用氯化汞作为组织学固定剂与环境大气中高浓度的汞蒸气（高达0 - 5纳摩尔/升）以及汞化合物（总汞达1 - 0纳摩尔/升）有关。暴露于这种环境的技术人员尿汞增加（中位数为265纳摩尔/24小时），蛋白质排出量也增加（中位数为117毫克蛋白质/24小时）。常规控制措施，如通风和小心处理氯化汞溶液，将大气汞蒸气水平降低到可接受的限度内（阈限值：烷基化合物为0 - 01毫克/立方米（0 - 05纳摩尔/升），除烷基外的所有形式为0 - 05毫克/立方米（0 - 25纳摩尔/升））。这种降低与技术人员尿液中微量蛋白尿的消失有关。应尽量减少组织学实验室被氯化汞污染的情况。
