Okusa M, Shiraishi T, Kubo T, Matsunaga T
Department of Otolaryngology, Osaka University Medical School, Japan.
Acta Otolaryngol Suppl. 1993;501:54-8. doi: 10.3109/00016489309126215.
In 62 patients, whose hearing acuity was normal or profoundly impaired, tinnitus suppression was attempted through electrical promontory stimulation (EPS) with a Promontory Stimulator (Cochlear Co.). The causes of tinnitus were idiopathic sudden deafness, ototoxicity, noise induced hearing loss, labyrinthitis, Meniere's disease, acoustic neuroma and unknown origin. Reduction of tinnitus was achieved in 46 of 68 ears (67.6%). Of these, EPS was most effective in cases of noise induced hearing loss (100.0%; 3/3), followed by those of idiopathic sudden deafness (87.5%; 14/16), Meniere's disease (83.3%; 5/6), labyrinthitis (75.0%; 6/8), ototoxicity (66.6%; 4/6) and unknown origin (76.4%; 13/17). The treatment had hardly any effect on patients with acoustic neuroma (8.3%; 1/12). Twenty patients reported residual inhibition with a duration ranging from several hours to one week. Our results suggest that tinnitus due to cochlear lesions can be suppressed by EPS. This technique may also be useful for differentiation between tinnitus resulting from cochlear lesions and from retrocochlear lesions.
在62例听力正常或严重受损的患者中，尝试使用岬刺激器（科利耳公司）通过电刺激岬（EPS）来抑制耳鸣。耳鸣的病因包括特发性突聋、耳毒性、噪声性听力损失、迷路炎、梅尼埃病、听神经瘤及病因不明。68只耳中有46只（67.6%）耳鸣减轻。其中，EPS对噪声性听力损失患者最为有效（100.0%；3/3），其次是特发性突聋患者（87.5%；14/16）、梅尼埃病患者（83.3%；5/6）、迷路炎患者（75.0%；6/8）、耳毒性患者（66.6%；4/6）及病因不明患者（76.4%；13/17）。该治疗对听神经瘤患者几乎没有效果（8.3%；1/12）。20例患者报告有残余抑制，持续时间从数小时至1周不等。我们的结果表明，耳蜗病变所致耳鸣可通过EPS得到抑制。该技术可能也有助于鉴别耳蜗病变与蜗后病变所致的耳鸣。
