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猪直肠狭窄的发病机制。I. 自然发生的疾病及其与沙门氏菌病的关联。

The pathogenesis of porcine rectal stricture. I. The naturally occurring disease and its association with salmonellosis.

作者信息

Wilcock B P, Olander H J

出版信息

Vet Pathol. 1977 Jan;14(1):36-42. doi: 10.1177/030098587701400105.

Abstract

Rectal stricture occurred in each of 23 pigs submitted for necropsy from eight Indiana farms. Each stricture was an annular cicatrization of the rectal wall, 2.0-5.0 cm anterior to the anorectal junction. Emaciation, colonic dilatation and compression atrophy of abdominal and thoracic viscera were a result of chronic obstipation and inanition. Most pigs with strictures had severe enteric disease 4-8 weeks before the strictures occurred. Salmonella typhimurium was isolated from seven of the eight groups of pigs examined. Ulcerative proctitis, a possible precursor of rectal stricture, was frequently in pigs with enterocolitis caused by S. typhimurium.

摘要

来自印第安纳州八个农场送检尸检的23头猪均出现直肠狭窄。每个狭窄均为直肠壁的环形瘢痕形成,位于肛门直肠交界处前方2.0 - 5.0厘米处。消瘦、结肠扩张以及腹部和胸部脏器的压迫性萎缩是慢性便秘和营养不良的结果。大多数出现狭窄的猪在狭窄发生前4 - 8周患有严重的肠道疾病。在所检查的八组猪中,有七组分离出了鼠伤寒沙门氏菌。溃疡性直肠炎是直肠狭窄的一个可能先兆,在由鼠伤寒沙门氏菌引起的小肠结肠炎猪中经常出现。

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